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delirium/infarzierung

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Seite 1 von 266 Ergebnisse

Delirium following acute myocardial infarction: incidence, clinical profiles, and predictors.

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OBJECTIVE To examine the incidence, clinical profile, and predictors of delirium following acute myocardial infarction (MI). METHODS The study sample included 212 consecutive patients with acute MI who were admitted to the coronary intensive care unit of a university hospital. RESULTS Delirium was

Intravascular large B-cell lymphoma presenting as acute hemorrhagic cerebral infarct with delirium.

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Intravascular large B-cell lymphoma (IVLBCL) is a cumbersome diagnosis to make in vivo, particularly because of its elusive nature and ability to be a relatively nonspecific 'great mimicker'. Although it frequently has skin manifestations, it often escapes diagnosis due to its angiotrophism and

Slow progressive bilateral posterior artery infarction presenting as agitated delirium, complicated with Anton's syndrome.

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Three patients presented with an acute agitated delirium as the earliest sign of bilateral posterior cerebral artery infarction. All patients showed a unique slow progressive deterioration with a remarkably long interval between the first neuropsychological and subsequent visual and neurological

Pre-existing cerebral infarcts as a risk factor for delirium after coronary artery bypass graft surgery.

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OBJECTIVE Delirium is a common and critical clinical syndrome in older patients. We examined whether abnormalities in the brain that could be assessed by magnetic resonance imaging predisposed patients to develop delirium after coronary artery bypass graft surgery. We also analysed the association

Manic delirium and frontal-like syndrome with paramedian infarction of the right thalamus.

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A disinhibition syndrome affecting speech (with logorrhoea, delirium, jokes, laughs, inappropriate comments, extraordinary confabulations), was the main manifestation of a right-sided thalamic infarct involving the dorsomedian nucleus, intralaminar nuclei and medial part of the ventral lateral
Acute confusional state (ACS) and acute agitated delirium (AAD) after infarction in the right middle cerebral artery territory were investigated in 41 consecutive patients. Acute confusional state was diagnosed on the basis of the results of the Mini-Mental State Examination, and AAD was

Syndrome of agitated delirium and visual impairment: a manifestation of medial temporo-occipital infarction.

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Three patients presented with sudden visual impairment followed by agitated delirium one to three days later. Examination revealed marked agitation, dementia, and loss of vision. Computerised axial tomography demonstrated temporo-occipital infarctions. All recovered from the agitated state in four

Acute confusional states with right middle cerebral artery infarctions.

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Three patients presenting predominantly with acute confusional states (ACS) are shown to have infarctions in the distribution of the right middle cerebral artery. It is suggested that the main deficit in ACS is in the function of selective attention. On the basis of cortical connections of

[An unusual sequela of a frequently occurring neurologic disorder: delirium caused by brain infarct].

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Six patients are described with delirium after cerebral infarction. Five had a right-sided parietal infarction with involvement of the inferior parietal lobule. One patient presented with a right-sided medial temporo-occipital infarction. The mild neurological signs were dominated by the delirium.

Elderly patient with delirium after myocardial infarction.

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Delirium is a transient global disorder of cognition. Almost any medical illness or medication can cause delirium. Here, we report a 71-year-old male who presented to the emergency department with a sudden change in mental status, which later resolved. An electrocardiogram was consistent with acute
The present prospective study aimed to investigate the incidence and risk factors of delirium after primary percutaneous coronary intervention (PCI) in older adults with acute ST-segment elevation myocardial infarction (STEMI). A total of 111 patients (age, ≥65 years) with acute STEMI following

Confusion or delirium in patients with posterior cerebral arterial infarction.

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OBJECTIVE To identify the possible anatomic sites and risk factors for the development of confusion or delirium in patients with posterior cerebral arterial (PCA) infarction. METHODS Twenty-nine patients aged 34-86 years with PCA infarction were divided into two groups: one with and the other

Acute myocardical infarction due to delirium tremens.

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A 36-year-old patient with normal-appearing coronary arteries suffered an acute Q-wave myocardial infarction during acute alcohol withdrawal and delirium tremens. Sympathetic hyperactivity with coronary spasm and increased platelet reactivity are probably the underlying mechanisms.

Repetitive myocardial infarctions secondary to delirium tremens.

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Delirium tremens develops in a minority of patients undergoing acute alcohol withdrawal; however, that minority is vulnerable to significant morbidity and mortality. Historically, benzodiazepines are given intravenously to control withdrawal symptoms, although occasionally a more substantial

Acute confusional states secondary to infarctions in the territory of the posterior cerebral artery in elderly patients.

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Acute confusional states and agitated delirium are among the most common psychopathologic disorders in the elderly. However, they are rarely reported in the course of infarcts in the territory of the posterior cerebral artery. This study involving thirteen patients aged more than 65 years suggests
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