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hypersensitivity/− nicotin
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Seite 1 von 1300 Ergebnisse
Environmental mycological study and allergic respiratory disease among tobacco processing workers.
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This study presents the results of an investigation of respiratory symptoms, lung function, chest X-ray examinations, analysis of specific IgE antibodies and skin prick tests to fungi on 130 tobacco-processing workers and 112 control workers. Industrial hygiene survey and environmental mycological
The Prevalence of Allergic Rhinitis and It's Relationship With Second-Hand Tobacco Smoke Among Adults in Iran.
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To determine the prevalence of allergic rhinitis (AR) among adult residents and investigate whether second-hand tobacco smoke (SHS) is correlated with AR as one of the global health problems and one of the most common respiratory disorders reducing the quality of life. A telephone interview survey
Challenge with environmental tobacco smoke exacerbates allergic airway disease in human beings.
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BACKGROUND
Despite widespread perceptions that environmental tobacco smoke (ETS) is a potent risk factor for allergic airway disease, epidemiologic studies studying this have been equivocal. There is a clear need for experimental studies to address these questions.
OBJECTIVE
We directly tested the
Prenatal nicotine exposure induces HPA axis-hypersensitivity in offspring rats via the intrauterine programming of up-regulation of hippocampal GAD67.
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The intrauterine programming of hypothalamic-pituitary-adrenal (HPA) axis hypersensitivity is associated with chronic adult diseases. Our previous studies demonstrated the HPA-axis hypersensitivity in offspring rats induced by prenatal nicotine exposure. The goal of the present study is to further
The association between pre- and postnatal tobacco smoke exposure and allergic sensitization during early childhood. Multicentre Allergy Study Group, Germany.
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OBJECTIVE
of our study was to investigate the effect of pre- and postnatal passive tobacco smoke exposure on the incidence of allergic sensitization.
METHODS
Specific sensitization to food, outdoor and indoor allergens was determined in 342 children at the age of 1, 2 and 3 years. Parents were asked
Allergic sensitization, rhinitis, and tobacco smoke exposure in U.S. children and adolescents.
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BACKGROUND
Childhood tobacco exposure has been linked with sinonasal pathology, and may be associated with allergic sensitization. This study evaluates the association between exposure to active smoking or secondhand smoke (SHS) and the prevalence of rhinitis and allergic sensitization in the U.S.
Influence of exposure to tobacco cigarette smoke on the eosinophil count in the nasal mucosa of young patients with perennial allergic rhinitis.
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BACKGROUND
To assess the influence of exposure to tobacco cigarette smoke on the eosinophil count and the frequency of apoptosis of eosinophils in the nasal mucosa of teenagers with perennial allergic rhinitis.
METHODS
Fifty patients were evaluated (aged 10 to 19 years old): 25 patients with and 25
Rhinomanometry in young patients with perennial allergic rhinitis with/without recent exposure to tobacco smoke.
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OBJECTIVE
To assess the influence of environmental exposure to tobacco smoke on the nasal symptoms and nasal resistance of young patients with perennial allergic rhinitis.
METHODS
Cross-sectional study.
METHODS
Tertiary referral centre.
METHODS
Fifty patients were evaluated (aged 10-19 years old):
Influence of environmental tobacco smoke on asthma in nonallergic and allergic children.
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The relation between exposure to environmental tobacco smoke and childhood asthma is not clear. A 1993 study of 892 subjects age 6-17 years (87.5% of 1,019 eligible subjects) living in Humboldt, Saskatchewan, showed that a lifetime history of asthma and asthma attack during the past 12 months were
[Anti-nicotine education applied in relation of parents of the diseased children on chronic allergic diseases of respiratory system].
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The allergies of respiratory system are at children the frequent illnesses. Among favorable them factors, risk on passive smoking tobacco can be also. Passive smoking is defined as risk non-smoking on tobacco smoke in environment. Recent reports represent that smoking in home environment tobacco
Tobacco hypersensitivity and environmental tobacco smoke exposure in a pediatric population.
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BACKGROUND
Skin testing and RAST have verified the existence of tobacco-specific IgE. However, published studies report conflicting results concerning the clinical significance of tobacco IgE. Previous studies have not focused on the role of environmental tobacco smoke (ETS) as it relates to tobacco
Relationship of upper airway disease to tobacco smoking and allergic markers: a cohort study of men followed up for 5 years.
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Data derived from a cohort study of 191 men, seen 5 years apart, were used to investigate the involvement in allergic as well as in nonallergic upper airway disease (UAD) of two risk factors, immediate hypersensitivity and tobacco smoking, the roles of which have been well established in lower
[Associations between family history of allergy, exposure to tobacco smoke, active smoking, obesity, and asthma in adolescents].
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OBJECTIVE
The relationships between asthma in adolescents and various environmental and social exposures needs to be clarified. The aim of this study was to determine the association between family history of allergy, passive or active tobacco smoking, obesity, and asthma in adolescents.
METHODS
A
[Respiratory and allergic diseases of children : Temporal trends, urban-rural differences, and in association with environmental tobacco smoke exposure].
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OBJECTIVE
Ten years after the establishment of health monitoring units (GME) in Bavaria, temporal trends and urban-rural differences in parent-reported respiratory and allergic diseases as well as environmental tobacco smoke (ETS) exposure in preschoolers were analyzed in an explorative manner.
Environmental tobacco smoke and progesterone alter lung inflammation and mucous metaplasia in a mouse model of allergic airway disease.
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The prevalence and severity of asthma is sexually dimorphic. Adult women have a higher incidence of asthma than men. This suggests that this disease may have a hormonal component. Progesterone has been shown to elicit an immune response similar to that seen in allergic asthma and previous studies
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