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hyperthyroidism/kopfschmerz

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A rare cause of headache: cerebral venous sinus thrombosis due to hyperthyroidism.

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Headache represents up to 4% of all emergency department (ED) visits. Emergency physicians generally are concerned with identifying those patients whose headaches are caused by life-threatening conditions. Cerebral venous sinus thrombosis may be difficult to diagnose clinically because of its

Hyperthyroidism presenting with persistent vomiting, headache and deranged liver function tests.

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[Headache secondary to hyperthyroidism].

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[Dihydroergotamine in the headache of hyperthyroid patients].

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Hyperthyroidism and thrombophilia in cerebral arterial and venous thrombosis: a case report and critical review.

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BACKGROUND People with hyperthyroidism, thrombophilia, or oral contraceptive (OC) use are reported to have a risk in developing arterial or venous thrombosis. There has been a rare case reporting concurrent arterial and venous thrombosis in such patients. METHODS We presented a 44-year-old woman

Thyrotoxicosis presenting with headache.

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We report three cases of thyrotoxicosis who presented acutely with headache to our neurology service in a 1-year period. In two of these patients there was a pre-existing or subsequent history of migraine. With hindsight, there were other clinical features of thyrotoxicosis but this diagnosis had

[Monosymptomatic hyperthyroidism and TSH-producing adenoma: successful therapy with octreotide].

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METHODS Magnetic resonance imaging (MRI) of the central nervous system was performed on a 72-year-old woman who was hyperthyroid without suppression of the thyroid-stimulating hormone (TSH) and had complained of a recent onset of headaches. MRI demonstrated a space-occupying lesion, 1 cm in

Thyroid function in patients with chronic headache.

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Headache is one of the neurological manifestations of hypothyroidism but it is unknown whether there is a relationship between hyperthyroidism and chronic headache. Thyroid function tests were performed in 30 patients with chronic headache. Six were found to have hyperthyroidism and none had

Cerebral venous thrombosis with auto-immune hyperthyroidism.

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Sudden deterioration of visual functions warrants comprehensive ophthalmic examination with evaluation for systemic association. Cerebral venous thrombosis (CVT) is an uncommon disorder that can present with neurological deficits. We report a young female patient aged 28 years who presented with

A Case of Cerebral Venous Thrombosis and Deep Venous Thrombosis Due to Hyperthyroidism with Increased Factor VIII Activity.

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A 48-year-old woman was admitted to our hospital because of headache and fever. She was diagnosed with aseptic meningitis. Five days later, she had a seizure and developed left hemiparesis. Magnetic resonance imaging showed hyperintensity in the right parietal area on fluid attenuated inversion

TSH-induced hyperthyroidism caused by a pituitary tumor.

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BACKGROUND A 45-year-old man presented with frontal headache and visual disturbances to our clinic. For the previous 5 years, he had been receiving treatment for long-lasting mild hyperthyroidism with antithyroid therapy, but therapy had not been carefully followed. During the last 2 years he had

Use of beta-adrenoceptor blocking drugs in hyperthyroidism.

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There is an increasing use and variety of beta-adrenoceptor blocking agents (beta-blockers) available for the treatment of hyperthyroidism. Recent comparative studies suggest that atenolol (200mg daily), metoprolol (200mg daily); acebutolol (400mg daily), oxprenolol ( 160mg daily), nadolol ( 80mg
BACKGROUND Radioactive iodine (I) (RAI) is used widely for the treatment of hyperthyroidism either as a first-line treatment or following relapse after antithyroid drug treatment. Intrathyroidal retention of RAI is considered an important determinant of its effectiveness, which is believed to be

Hyperthyroidism as a cause of autonomic dysreflexia.

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A 25-yr-old female tetraplegic patient experienced autonomic dysreflexia episodes involving hypertension, headache, facial flushing, and tachycardia. The symptoms were not related to the bladder or bowel. The episodes did not seem to be linked to any mechanical cause. The patient was incidentally
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