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n acetyl beta d glucosaminidase/nekrose
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Seite 1 von 154 Ergebnisse
Urinary N-acetyl-beta-D-glucosaminidase and neopterin aid in the diagnosis of rejection and acute tubular necrosis in initially nonfunctioning kidney grafts.
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OBJECTIVE
The study aimed at investigating urinary neopterin, a marker of cellular immune response, and urinary N-acetyl-beta-D-glucosaminidase (NAG), a marker of tubular damage, as noninvasive means to differentiate between acute tubular necrosis (ATN) and rejection in initially nonfunctioning
The activity of N-acetyl-beta-D-glucosaminidase and tumor necrosis factor-alpha at early stage of diabetic retinopathy development in type 1 diabetes mellitus children.
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OBJECTIVE
The assessment of the clinical significance of TNF-alpha, IL-10 and NAG with its A and B isoforms concentrations in children with DM type 1 for the detection of early stages of both diabetic retinopathy and nephropathy.
METHODS
One hundred and two children with DM type 1 and 35 healthy
Occupational exposure to chlorinated organic solvents and its effect on the renal excretion of N-acetyl-beta-D-glucosaminidase.
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Organic chlorinated hydrocarbon solvents are known to be nephrotoxic. However, very little is known about renal integrity after occupational exposure to these solvents. Increased urinary N-acetyl-beta-D-glucosaminidase (NAG) excretion caused by necrosis of renal tubular cells can be used as a marker
The clinical value of urinary N-acetyl-beta-D-glucosaminidase levels in childhood age group.
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N-acetyl-beta-D-glucosaminidase is a high molecular-weight lysosomal enzyme found in many tissues of the body. It cannot pass into glomerular ultrafiltrate due to its high molecular weight. However, this enzyme shows high activity in renal proximal tubular cells, and leaks into the tubular fluid as
Nephroprotective effects of subcapsular transplantation of metanephric mesenchymal cells on gentamicin-induced acute tubular necrosis in rats.
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BACKGROUND
The subcapsular transplantation of metanephric mesenchymal cells (MMCs) may be a new therapeutic approach for the treatment of acute tubular necrosis (ATN). To investigate this hypothesis and provide evidence for its possible use in the clinic, we evaluated the nephroprotective effects of
Indices of urine N-acetyl-beta-D-glucosaminidase and gamma-glutamyl transpeptidase activities in clinically normal adult dogs.
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OBJECTIVE
To establish reference ranges for indices of urine N-acetyl-B-D-glucosaminidase (NAG) and gamma-glutamyl transpeptidase (GGT) activities in clinically normal adult dogs.
METHODS
38 dogs.
METHODS
Each dog underwent a physical examination, CBC, serum biochemical analysis, urinalysis, and
Increased serum N-acetyl-beta-D-glucosaminidase activity in human hypertension.
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Compared to values obtained in healthy normotensive control subjects, serum activity of the lysosomal enzyme N-acetyl-beta-D-glucosaminidase (NAG) was found to be elevated in patients with untreated or treated essential hypertension and in patients with renovascular hypertension. Increased NAG
N-acetyl-B-D-glucosaminidase and inflammatory response after cardiopulmonary bypass.
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OBJECTIVE
To determine the changes in activity of plasma N-acetyl-beta-D-glucosaminidase, a marker for inflammation as well as renal, pulmonary and cardiac damage and proinflammatory cytokines in patients undergoing coronary artery bypass grafting and find out the relationship between their plasma
Clinical evaluation of urinary N-acetyl-beta-D-glucosaminidase activity in patients receiving aminoglycoside and cephalosporin drugs.
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N-acetyl-beta-D-glucosaminidase (NAG) activity in urine was measured as an indicator for detecting the onset of renal damage in patients receiving aminoglycoside and cephalosporin drugs. The studies reveal that gentamicin appears to be most nephrotoxic of the aminoglycoside antibiotics. Polyuria,
[Nephrotoxicity of aminoglycosides and cephalosporins. Value of examining the isoenzyme profile of N-acetyl-beta-D-glucosaminidase].
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It is a well known fact that aminoglycosides increase urinary action of N-acetyl-beta-D-glucosaminidase (NAG) but the significance of this fact remains, as yet, nuclear. The aim of this study is firstly to attempt to form an explanation of the enzymatic effects of aminoglycosides by studying the
Serum and urinary N-acetyl-beta-D-glucosaminidase activity among the inhabitants of a rural area in Japan--the effect of age and hypertension.
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In this paper, the normal values, distribution, and variations in serum and urinary activities of the lysosomal enzyme, N-acetyl-beta-D-glucosaminidase (NAG), were estimated in a rural area of Japan. The frequency distribution of NAG activities both in the serum and urine of 1,152 males and females
The effects of arginine, dextran and Haemaccel infusions on urinary albumin, beta 2-microglobulin and N-acetyl-beta-D-glucosaminidase.
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Male volunteers were infused with L-arginine dextran and Haemaccel. Arginine (0.5 g/kg body weight infused over 30 min) resulted in transient highly significant increases in urinary albumin (p less than 0.001), beta 2-microglobulin (p less than 0.001) and N-acetyl-beta-D-glucosaminidase [NAG] (p
Effect of infliximab, a tumor necrosis factor-alpha inhibitor, on doxorubicin-induced nephrotoxicity in rats.
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Treatment with the chemotherapeutic agent, doxorubicin (DOX), is limited by nephrotoxicity. We investigated the possible protective effect of infliximab, a tumor necrosis factor alpha (TNF-α) inhibitor on DOX-induced nephrotoxicity. Rats were treated with a single intraperitoneal (ip) injection of
Prognostic value of tubular proteinuria and enzymuria in nonoliguric acute tubular necrosis.
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BACKGROUND
Acute tubular necrosis (ATN) has high mortality, especially in patients who require renal replacement therapy (RRT). We prospectively studied the diagnostic accuracy of the urinary excretion of low-molecular-weight proteins and enzymes as predictors of a need for RRT in ATN.
METHODS
In 73
Assessment and prediction of acute kidney injury in patients with decompensated cirrhosis with serum cystatin C and urine N-acetyl-β-D-glucosaminidase.
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OBJECTIVE
For appropriate management of acute kidney injury (AKI) in cirrhotic patients, accurate differentiation of the types of AKI, prerenal azotemia (PRA), hepatorenal syndrome (HRS), and acute tubular necrosis (ATN) is very important. Urine N-acetyl-β-D-glucosaminidase (NAG) has been proposed
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