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scopolamine/infarzierung
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Seite 1 von 26 Ergebnisse
Effects of low dose transdermal scopolamine on heart rate variability in acute myocardial infarction.
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OBJECTIVE
We hypothesized that by enhancing parasympathetic activity, low dose transdermal scopolamine would increase heart rate variability after myocardial infarction.
BACKGROUND
Low heart rate variability is associated with increased mortality after acute myocardial
Scopolamine increases vagal tone and vagal reflexes in patients after myocardial infarction.
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OBJECTIVE
The goal of this study was to assess the hypothesis that transdermal scopolamine would increase vagal activity in patients after myocardial infarction.
BACKGROUND
In postmyocardial infarction patients, low heart rate variability and reduced baroreceptor reflex sensitivity are associated
Influence of transdermal scopolamine on cardiac sympathovagal interaction after acute myocardial infarction.
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In 41 survivors of acute myocardial infarction (AMI) a prospective study was performed in 2 sequential phases. In phase 1, the role of baroreflex sensitivity and heart rate variability as predictors of inducible and spontaneous sustained ventricular tachyarrhythmias was evaluated. In phase 2, the
Low dose transdermal scopolamine increases cardiac vagal tone in patients after acute myocardial infarction.
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OBJECTIVE
To investigate whether transdermal scopolamine increased cardiac vagal activity in patients during the acute phase of myocardial infarction.
METHODS
30 patients with a first acute myocardial infarction and preserved sinus rhythm who were on no drug that could influence the sinus node were
Low doses of scopolamine increase cardiac vagal tone in the acute phase of myocardial infarction.
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BACKGROUND
Reduced cardiac vagal tone in patients with myocardial infarction (MI) is associated with a high risk of sudden death. Muscarinic blocking agents in small doses induce a paradoxical increase in cardiac vagal activity in normal subjects. We tested whether low doses of scopolamine delivered
[Effects of scopolamine plus atropine on experimental myocardial infarction in rabbits].
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[A report on 4 cases of acute myocardial infarction complicated by refractory ventricular tachycardia successfully treated with scopolamine].
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Ionic channels, cholinergic mechanisms, and recovery of sensorimotor function after neocortical infarcts in rats.
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Unilateral photochemical infarcts were produced in the hind limb sensorimotor neocortex of 243 rats by intravenous injection of the fluorescein derivative Rose Bengal and focal illumination of the intact skull surface. Facial contact stimuli governed the degree and recovery rate of contralateral
Autonomic modulation during acute myocardial ischemia by low-dose pirenzepine in conscious dogs with a healed myocardial infarction: a comparison with beta-adrenergic blockade.
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Experimental and clinical evidence documents the beneficial effects of blocking sympathetic activity and modulating heart rate to reduce risk for lethal events in ischemic heart disease. Beside beta-adrenergic receptor blockade, vagal activation is a meaningful approach but not yet easily
Scopolamine improves autonomic balance in advanced congestive heart failure.
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BACKGROUND
Sympathetic hyperactivity and parasympathetic withdrawal in patients with congestive heart failure correlate closely with disease severity and overall survival. The modulating effects of drugs on the autonomic dysfunction may contribute to improve survival. Low-dose scopolamine has a
Do increases in markers of vagal activity imply protection from sudden death? The case of scopolamine.
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BACKGROUND
Low-dose scopolamine increases heart rate variability (HRV) in patients with a prior myocardial infarction (MI). This observation, combined with the evidence that elevated cardiac vagal activity during acute myocardial ischemia is antifibrillatory, has generated the hypothesis that
Effects of right-hemisphere cortical infarction and muscarinic acetylcholine receptor blockade on spatial visual attention performance in rats.
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The syndrome of hemispatial neglect is defined as an inability to report, respond or orient to stimuli contralateral to a cerebral lesion despite intact elementary sensory or motor function. This syndrome is typically observed after lesions of the right cerebral cortex, and has been associated with
Disordered cholinergic neurotransmission and dysautoregulation after acute cerebral infarction.
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The possible role of displaced neurotransmitter acetylcholine (ACHh) in dysautoregulation was examined after experimental regional cerebral infarction was produced by occluding the middle cerebral artery (MCA) in babons. Regional cerebral blood flow (rCBF) was measured after intracarotid injection
Urodynamics and bladder muscarinic receptors in rats with cerebral infarction and bladder outlet obstruction.
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We characterized muscarinic receptor binding and urodynamic parameters in rats with cerebral infarction and chronic bladder outlet obstruction as models of detrusor overactivity. Bladder weight showed little significant difference between the cerebral-infarcted and sham rats, but the bladder weight
Differential effects of scopolamine on neuronal survival in ischemia and glutamate neurotoxicity: relationships to the excessive vulnerability of the dorsoseptal hippocampus.
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The neurodegeneration in the CA1 subfield of hippocampus exhibited a dorsal-ventral gradient of susceptibility in global ischemia (82% dorsoseptally and only 16% ventrotemporally). Scopolamine (SCOP) did not improve the neuronal damage caused by the global ischemic challenge in rats and did not
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