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thrombophilia/hypoxie
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Hypoxia-reoxygenation contributes to increased frequency of venous thromboembolism in air travellers.
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Commercial air travel is widespread, with close to 2 billion people traveling each year. The association between venous thromboembolism (VTE) and confined sitting has been established and a correlation between the distance traveled and its incidence had been documented. Most studies underscore
Hypoxia Induces a Prothrombotic State Independently of the Physical Activity.
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Hypoxia (oxygen deprivation) is known to be associated with deep vein thrombosis and venous thromboembolism. We attempted to get a better comprehension of its mechanism by going to high altitude, thereby including the potential contributing role of physical activity. Two groups of 15 healthy
The role of hypoxia and platelets in air travel-related venous thromboembolism.
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Although somewhat controversial, there is good evidence that long-distance travel in general is a risk factor for venous thromboembolism, even in the absence of other risk factors. This is probably due to effects consequent to prolonged sitting but air travel in particular may be associated with
Is mild normobaric hypoxia a risk factor for venous thromboembolism?
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BACKGROUND
Modern air travel entails a cabin altitude between 1520 and 2440 m (5000-8000 ft) and thus exposure to mild hypoxia. There is debate as to whether hypoxia is causally related to venous thromboembolism (VTE) occurring during or after travel. One study suggested that a short period of
Enhanced platelet inhibition treatment improves hypoxemia in patients with severe Covid-19 and hypercoagulability. A case control, proof of concept study
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Patients affected by severe coronavirus induced disease-2019 (Covid-19) often experience hypoxemia due to alveolar involvement and endothelial dysfunction, which leads to the formation of micro thrombi in the pulmonary capillary vessels. Both hypoxemia and a prothrombotic diathesis have been
[Mechanism of spontaneous correction of hypercoagulability in severe short-term hypoxia].
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[Mechanism inducing blood hypercoagulability in severe hypoxia].
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Thrombolytic therapy for venous thromboembolism: current clinical practice.
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BACKGROUND
Venous thromboembolism (VTE) is a life-threatening condition for which thrombolytic therapy may be beneficial. The appropriate setting for the use of thrombolytic therapy remains controversial. More than 10 years ago we described the case-based practice patterns for the use of
Evaluation of hypercoagulability using soluble fibrin monomer complex in sick newborns.
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BACKGROUND
Soluble fibrin monomer complex (SFMC) and fibrin monomer (FM) are well known as markers for hypercoagulability, but such measurements have not been investigated in detail for the neonate. To identify the presence of a hypercoagulable state in sick newborns, the behavior of SFMC with
Is there a connection between long airplane flight, venous thromboembolism, and sleep-disordered breathing?
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Commercial passenger flights have been increasing around the world. The effect of these flights on health is unclear. Venous thromboembolism has been noted after recent long-distance airplane flight, even in the absence of other risk factors. Hypoxia caused by the low ambient pressure during flights
Operation for chronic pulmonary thromboembolism accompanied by thrombophilia in 8 patients.
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BACKGROUND
Medical therapy for chronic pulmonary thromboembolism is limited, and surgical treatment has become more frequent recently. We have performed pulmonary thromboendarterectomy on 8 patients with chronic pulmonary thromboembolism accompanied by thrombophilia.
METHODS
The patients were 6 men
Awareness Level of Airline Pilots on Flight-Associated Venous Thromboembolism
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INTRODUCTION: Studies have identified a great number of physiological conditions, including venous thromboembolism and hypoxia, that may give rise to medical disqualifications and in-flight incapacitations that can be costly to individuals and organizations. Over the past three decades, much
Thrombosis and thrombophilia: principles for pediatric patients.
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Thrombotic complications in pediatric patients are increasingly recognized due to increased use of invasive procedures, heightened awareness, improved imaging and prothrombotic lifestyle choices. Multiple risk factors are often present in pediatric patients with thrombosis. The most common risk
Intravascular coagulation activation in a murine model of thrombomodulin deficiency: effects of lesion size, age, and hypoxia on fibrin deposition.
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We consecutively inactivated both alleles of the thrombomodulin (TM) gene in murine embryonic stem (ES) cells and generated TM-deficient (TM-/-) chimeric mice. Quantitation of an ES-cell marker and protein C cofactor activity indicates that up to 50% of pulmonary endothelial cells are ES-cell
Effect of chronic hypoxia on fibronectin in plasma, bronchoalveolar lavage fluid and liver of rats.
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The present study was undertaken to measure fibronectin (FN) concentration in plasma and bronchoalveolar lavage fluid (BALF) with an enzyme linked immunosorbent assay (ELISA) method, to observe the distribution of FN in liver with an immunohistochemical stain method and to investigate the effect of
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