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uremia/phosphatase
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Seite 1 von 101 Ergebnisse
The effect of experimental uremia on potassium-activated phosphatase from erythrocyte and cardiac membranes.
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Unilateral nephrectomy and partial resection of the contralateral kidney in rats causes uremia that lasts for several weeks and influenced overall (Na+, K+)-ATPase activity as well as p-nitrophenyl phosphatase (p-NPPase) in the plasma membranes of erythrocytes and heart. Both enzymatic activities
[Function of the neutrophils in uremia - with special reference to neutrophil alkaline phosphatase activities, phagocytosis of charcoal particles and reduction of NBT].
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Persistence of Vascular Calcification after Reversal of Uremia.
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The extent to which vascular calcification is reversible and the possible mechanisms are unclear. To address this, calcified aortas from uremic mice were transplanted orthotopically into normal mice, and the calcium content, histology, and minerals of the allografts were compared with the
Effect of acute experimental uremia on the stability of liver lysosomal membranes.
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Acute uremia was induced in male Swiss albino mice by complete urethral ligation and the animals were sacrificed 2, 4-6, 24, and 48 h after operation. Sham-opeated animals (without the urethral ligation) were similarly treated. The blood urea levels of animals with total urinary tract obstruction
Role of dietary phosphorus and degree of uremia in the development of renal bone disease in rats.
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The remnant kidney rat model has been extensively used for the evaluation of bone changes due to uremia. The present study aimed to assess the effect of the dietary phosphorus availability and of the severity of renal failure on bone histomorphometric changes and various biochemical markers over
An evaluation of a kinetic acid phosphatase method.
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The authors have evaluated a new kinetic acid phosphatase method in which the substrate is alpha-naphthyl phosphate. The original claim that this substrate was highly specific for the prostatic isozyme has been strongly challenged. Therefore, large numbers of patients in the following groupings were
Tissue calcification and alkaline phosphatase activity in uraemic rats.
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The activity and localization of alkaline phosphatase activity (AP) in aorta and heart, and the incidence of calcifications in aorta, heart and kidney as well as cardial fibrosis were studied in uraemic rats treated with 1,25-dihydroxycholecalciferol (1,25-DCHH) and Nifedipine. 1,25-DHCC treatment
Rapid suppression of plasma alkaline phosphatase activity after renal transplantation in patients with osteodystrophy.
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Plasma alkaline phosphate activity is a important marker of increased skeletal turnover (both resorption and formation) and bone disease in uraemia, but its value after renal transplantation is uncertain. The rate of fall of alkaline phosphatase was compared in three groups of uraemic patients with
Enzyme cytochemistry of rat organs after uremia with special reference to proteases.
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Wistar rat organs and tissues were investigated after acute and chronic uremia using enzyme cytochemical means whereby special attention was paid to plasma membrane and lysosomal proteases. Heart muscle, pancreas, spleen, stomach, duodenum, jejunum, colon and skeletal muscle did not show any
Surgically induced uremia in rats. II: Osseous PTH-susceptible signaling systems as predictors of bone resorption.
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Predicting the course of parathormone (PTH)-elicited bone turnover in both humans and experimental rat models with moderate chronic uremia, using only standard clinical chemistry analyses, is often difficult. Consequently, rat bone from 1 + 2/3 nephrectomized animals, after 230 days of progressive
[The index of leukocyte alkaline phosphatase in patients with hemodialysis (author's transl)].
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The index of the leukocyte alkaline phosphatase activity (LAP-I) was evaluated in 15 patients suffering from chronic uremia immediately before and after hemodialysis, as well as 16 or 18 and 40 or 42 hours thereafter. Immediately following hemodialysis the LAP-I was significantly reduced in
[Severe facial malformation caused by secondary hyperparathyroidism in uremia: report of 4 cases].
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Objective: To investigate the etiology, characteristics and prevention of severe facial deformity in patients with uremia entering the dialysis stage. Methods: Four cases with uremia in the dialysis stage who presented with severe facial deformity between October 2011 and November 2018
Effects of chronic uremia on bone.
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Iliac crest bone biopsies in patients with chronic renal failure with severely disturbed renal function were measured with regard to osteoclast activity and active and inactive osteoid. In none of the 21 patients had hemodialysis been applied. In uremia cases, as earlier demonstrated in hemodialysis
Application of total parathyroidectomy with auto-transplantation for uremia secondary hyperparathyroidism treatment.
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BACKGROUND
Secondary hyperparathyroidism (sHPT) is a common acquired disorder in patients with chronic renal failure. Despite the development of new therapeutic agents, a majority of patients will require parathyroidectomy. The aim of this study was to evaluate total parathyroidectomy with
Parathyroid and bone response of the diabetic patient to uremia.
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Biochemical and radiologic indices of bone disease were assessed in 26 insulin-dependent diabetic patients and 28 nondiabetic patients with endstage kidney disease. The two groups were comparable in age, sex, duration of renal failure, and length of time on dialysis. Diabetic patients showed
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