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eosinophilia/παχυσαρκία
Ο σύνδεσμος αποθηκεύεται στο πρόχειρο
Σελίδα 1 από 61 Αποτελέσματα
Obesity lowers the threshold of allergic sensitization and augments airway eosinophilia in a mouse model of asthma.
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Σύνδεση εγγραφή
BACKGROUND
Clinical and epidemiological studies show a close association between obesity and the risk of asthma development. The underlying cause-effect relationship between metabolism, innate and adaptive immunity, and inflammation remains to be elucidated.
METHODS
We developed an animal model to
Obesity and hiatal hernia may be non-allergic risk factors for esophageal eosinophilia in Japanese adults.
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Elevated sputum interleukin-5 and submucosal eosinophilia in obese individuals with severe asthma.
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BACKGROUND
The relationship between airway inflammation and obesity in severe asthma is poorly understood.
OBJECTIVE
We sought to determine the relationship between sputum mediator profiles and the distribution of eosinophilic inflammation and obesity in people with severe asthma.
METHODS
Clinical
Reply: Elevated sputum IL-5 and submucosal eosinophilia in obese individuals with severe asthma.
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Esophageal eosinophilia after radiofrequency ablation for Barrett's esophagus.
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Radiofrequency ablation (RFA) with HALO system has been developed as a new treatment option for Barrett's esophagus (BE). It had been observed that some patients had esophageal eosinophilia (EE) infiltration after RFA. The incidence and features of EE after RFA were systematically determined. From a
[Exacerbations in perimenstrual asthma. Clinical significance of peripheral blood eosinophilia and BMI].
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BACKGROUND
Asthma is a heterogeneous disease characterized by lower airways' obstruction, caused by various factors. There are many asthma phenotypes. Lately, perimenstrual asthma (PMA) with a pattern of exacerbations before and during menstruation as well as obesity associated asthma have been a
Obese asthmatic patients have decreased surfactant protein A levels: Mechanisms and implications.
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BACKGROUND
Eosinophils are prominent in some patients with asthma and are increased in the submucosa in a subgroup of obese patients with asthma (OAs). Surfactant protein A (SP-A) modulates host responses to infectious and environmental insults.
OBJECTIVE
We sought to determine whether SP-A levels
Cardiolipin profiles as a potential biomarker of mitochondrial health in diet-induced obese mice subjected to exercise, diet-restriction and ephedrine treatment.
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Cardiolipin (CL) is crucial for mitochondrial energy metabolism and structural integrity. Alterations in CL quantity or CL species have been associated with mitochondrial dysfunction in several pathological conditions and diseases, including mitochondrial dysfunction-related compound attrition and
Eosinophilia-myalgia syndrome case-associated contaminants in commercially available 5-hydroxytryptophan.
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Recently, 5-hydroxy-L-tryptophan (5-OHTrp) has been promoted as an alternative to banned L-tryptophan as a dietary supplement. It has been claimed to help alleviate obesity, insomnia, depression, and headaches. However, eosinophilia-myalgia syndrome (EMS)-like symptoms have also been associated with
Differential effects of obesity on eosinophilic vs. non-eosinophilic asthma subtypes.
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OBJECTIVE
Asthma is a heterogeneous disease composed of multiple disease subtypes. Obesity may worsen asthma, although the mechanism is poorly understood and its effects on different subtypes are not well characterized. We sought to determine whether obesity affects eosinophilic asthma differently
Cluster analysis of the phenotype of asthma and obesity.
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OBJECTIVE
Introduction: The combination of asthma and obesity can affect the mutual encumbrance to which other pathogenetic mechanisms join, which worsen the course of both diseases. The aim of the work is to analyze the features of the genotype and phenotype in patients with a combined course of
Scleroderma, fasciitis, and eosinophilia associated with the ingestion of tryptophan.
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An association between the ingestion tryptophan and a syndrome characterized by scleroderma-like skin abnormalities, fasciitis, and eosinophilia has recently been recognized in the United States. We report the clinical and histopathological findings in nine patients and the results of biochemical
Ciprofibrate, clofibric acid and respective glycinate derivatives. Effects of a four-week treatment on male lean and obese Zucker rats.
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Fibrates are widely prescribed in hyperlpidemic patients to prevent atherosclerosis. Their therapeutic use, however, can be associated with adverse effects like gastrointestinal disorders, myalgia, myositis and hepatotoxicity. In rodents large doses can even cause hepatocellular carcinoma.
Innate lymphoid cells contribute to allergic airway disease exacerbation by obesity.
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Epidemiologic and clinical observations identify obesity as an important risk factor for asthma exacerbation, but the underlying mechanisms remain poorly understood. Type 2 innate lymphoid cells (ILC2s) and type 3 innate lymphoid cells (ILC3s) have been implicated, respectively, in asthma and
IL-17 protein levels in both induced sputum and plasma are increased in stable but not acute asthma individuals with obesity.
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Obesity worsens asthma control partly through enhanced airway neutrophilia, altered lung mechanics and comorbidities, including obstructive sleep apnea syndrome, gastroesophageal reflux disease and depression. Although controversial, obesity may also cause poorer outcomes in acute asthma. IL-17 is
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