paresis/αιμορραγία

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Hemiparesis following post-partum haemorrhage and eclampsia.

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A primigravida at 41+ weeks gestation presented with spontaneous rupture of membranes. Labour was induced and later an emergency caesarean section was performed for failure to progress. The patient suffered a per-operative uterine tear and post-partum haemorrhage and required postoperative

Ataxic hemiparesis following thalamic hemorrhage: a case report.

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A 66-year-old man suddenly developed right ataxic hemiparesis. Sensations, somatosensory-evoked potentials and visual-evoked potentials were normal. Magnetic resonance imaging and computed tomography of the head showed an acute hemorrhage mainly in the ventrolateral nucleus of the left thalamus.

Hemorrhagic arachnoid cyst with third nerve paresis: CT and MR findings.

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We report the CT and MR appearance of a nontraumatic hemorrhagic arachnoid cyst presenting with a third nerve paresis in a 37-year-old man. The cyst, located in the left suprasellar area, contained a fluid-blood level with stigmata of subacute hemorrhage on both CT and MR studies.

Lower extremity monoparesis after aneurysmal subarachnoid hemorrhage.

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Two patients presented with acute subarachnoid hemorrhage from a ruptured intracranial aneurysm. Both patients were treated via endovascular coil embolization, and both developed delayed lower extremity monoparesis without associated symptoms that resolved over the ensuing months. An extensive

Outcome assessment of hemiparesis due to intracerebral hemorrhage using diffusion tensor fractional anisotropy.

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BACKGROUND This study aimed to evaluate the prognostic efficacy of magnetic resonance diffusion tensor fractional anisotropy (FA) for patients with hemiparesis due to intracerebral hemorrhage. METHODS Diffusion tensor FA brain images were acquired 14-21 days after putaminal and/or thalamic

Ipsilateral hemiparesis after putaminal hemorrhage due to uncrossed pyramidal tract.

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OBJECTIVE Previous case reports supported the presence of the uncrossed pyramidal tract in exceptional patients. However, most of these case reports have not fully discussed involvement of the motor cortex controlling the ipsilateral limbs. METHODS The authors investigated a 62-year-old man who

Ipsilateral hemiparesis caused by putaminal hemorrhage in a patient with horizontal gaze palsy with progressive scoliosis: a case report.

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BACKGROUND Horizontal gaze palsy with progressive scoliosis (HGPPS) is an autosomal recessive disorder caused by mutations in the ROBO3 gene, resulting in a critical absence of crossing fibers in the brainstem. METHODS We present a patient with ipsilateral hemiparesis caused by putaminal hemorrhage

Ataxic hemiparesis from small capsular hemorrhage. Computed tomography and somatosensory evoked potentials.

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We studied three cases that fit the clinical syndrome of ataxic hemiparesis. Computed tomography revealed small hemorrhages of the posterior limb of the internal capsule in all the cases, and somatosensory evoked potential studies predicted a disturbance of the lemniscal pathway from the thalamus to

Abducens nerve pareses associated with aneurysmal subarachnoid hemorrhage. Incidence and clinical features.

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BACKGROUND This study was performed to reveal the incidence and the etiology of abducens nerve pareses associated with aneurysmal subarachnoid hemorrhage. METHODS At the time of admission, CT scan was carried out, and the thickness of the prepontine subarachnoid clot was measured. RESULTS In total

Ataxic hemiparesis in patients with primary pontine hemorrhage.

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Two patients with small primary pontine hemorrhage developed a syndrome identical to ataxic hemiparesis, one of the lacunar syndromes of Fisher. The possible mechanisms of the homolateral cerebellar signs, pyramidal signs, and dysarthria are discussed. Lesions of the ipsilateral pontine nuclei may

Hypertensive putaminal hemorrhage presenting as pure motor hemiparesis.

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A 44 year old hypertensive man presented with a pure motor hemiparesis, and CT scan showed a putaminal hemorrhage. The clinical course was characterized by rapid resolution of the deficits. This case illustrates a variety of putaminal hemorrhage of good functional and vital prognosis, and stresses

Hypesthetic-ataxic-hemiparesis in thalamic hemorrhage.

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Acute onset hypesthetic-ataxic-hemiparesis is described in two hypertensive patients. Computed tomography (CT) showed an area of increased attenuation consistent with blood in contralateral thalamus. The pathophysiologic implications of the cerebellar and pyramidal system in thalamic hemorrhage is

The localization of central pattern generators for swallowing in humans--a clinical-anatomical study on patients with unilateral paresis of the vagal nerve, Avellis' syndrome, Wallenberg's syndrome, posterior fossa tumours and cerebellar hemorrhage.

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BACKGROUND Our understanding of brainstem swallowing centers is mainly based on experimental animals. In order to solve this problem also in humans, a clinical-anatomical study on dysphagic patients with different lesion patterns was performed. METHODS We studied 43 consecutively admitted dysphagic

Pure motor hemiparesis due to hypertensive putaminal haemorrhage.

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A hypertensive woman presenting as pure motor hemiparesis with rapid and complete recovery from the neurological deficit is described. The probable aetiology was primary hypertensive putaminal haemorrhage, as revealed by CT scan. This rare presentation has been described only once earlier and

Hemiparesis without responsible hematomas in patients with subarachnoid hemorrhage undergoing early aneurysmal repair.

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OBJECTIVE The presence of hemiparesis on arrival in patients with subarachnoid hemorrhage (SAH) is presumed to affect their prognosis, and intracranial hematomas with a mass effect responsible for the hemiparesis are frequently observed in these patients. The purpose of this study was to clarify the

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