adenosine/fatigue

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Muscle contraction and fatigue. The role of adenosine 5'-diphosphate and inorganic phosphate.

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Though many explanations are offered for the fatigue process in contracting skeletal muscle (both central and peripheral factors), none completely explain the decline in force production capability because fatigue is specific to the activity being performed. However, one needs to look no further

Randomized clinical trial on the effects of adenosine 5'-triphosphate infusions on quality of life, functional status, and fatigue in preterminal cancer patients.

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BACKGROUND One potential agent to improve symptoms and quality of life (QoL) in advanced cancer patients is adenosine 5'-triphosphate (ATP). Several reports suggest that ATP may positively affect QoL and survival in patients with advanced non-small-cell lung cancer. OBJECTIVE To investigate the

Central nervous system effects of caffeine and adenosine on fatigue.

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Caffeine ingestion can delay fatigue during exercise, but the mechanisms remain elusive. This study was designed to test the hypothesis that blockade of central nervous system (CNS) adenosine receptors may explain the beneficial effect of caffeine on fatigue. Initial experiments were done to confirm

Adenosine-5'-triphosphate (ATP) supplementation improves low peak muscle torque and torque fatigue during repeated high intensity exercise sets.

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BACKGROUND Intracellular concentrations of adenosine-5'-triphosphate (ATP) are many times greater than extracellular concentrations (1-10 mM versus 10-100 nM, respectively) and cellular release of ATP is tightly controlled. Transient rises in extracellular ATP and its metabolite adenosine have

[Treatment of chronic fatigue by adenosine triphosphoric acid].

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[ON THE USE OF ADENOSINE TRIPHOSPHORIC ACID AND OF COCARBOXYLASE IN PILOTS WITH SLIGHT OR INITIAL FLIGHT FATIGUE].

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[Effect of muscle load and fatigue on the adenosine triphosphate and creatine phosphate level in the skeletal muscles of the mackerel Trachurus mediterraneus].

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Time response of increases in ATP and muscle resistance to fatigue after low-level laser (light) therapy (LLLT) in mice.

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Recently, low-level laser (light) therapy has been used to increase muscle performance in intense exercises. However, there is a lack of understanding of the time response of muscles to light therapy. The first purpose of this study was to determine the time response for light-emitting diode therapy

Skeletal muscle energy metabolism and fatigue during intense exercise in man.

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Adenosine triphosphate (ATP) is the sole fuel for muscle contraction. During near maximal intense exercise the muscle store of ATP will be depleted in < 1s, therefore, to maintain normal contractile function ATP must be continually resynthesized. During intense exercise (from approximately 75% VO2

Metabolic abnormalities in chronic fatigue syndrome/myalgic encephalomyelitis: a mini-review.

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Chronic fatigue syndrome (CFS), commonly known as myalgic encephalomyelitis (ME), is a debilitating disease of unknown etiology. CFS/ME is a heterogeneous disease associated with a myriad of symptoms but with severe, prolonged fatigue as the core symptom associated with the disease. There are

Endurance exercise-induced and mental fatigue and the brain.

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It appears that the noradrenergic neurotransmitter system hastens central fatigue during prolonged exercise, a finding that coincides with a faster rate of increase in the rating of perceived exertion. Mental fatigue will affect several neurotransmitter systems, with presumably an important role for

Optimization of solid state culture conditions for the production of adenosine, cordycepin, and D-mannitol in fruiting bodies of medicinal caterpillar fungus Cordyceps militaris (L.:Fr.) Link (Ascomycetes).

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In general, Cordyceps sinensis is much more popular than C. militaris, though both species contain quite similar bioactive ingredients and exhibit medicinal activities. Many bioactive ingredients have been isolated from C. militaris, such as adenosine, cordycepin, D-mannitol, and exopolysaccharides.

Adenosine A2A receptor and TNF-α regulate the circadian machinery of the human monocytic THP-1 cells.

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Morning stiffness and increased symptoms of inflammatory arthritis are among the most common manifestations of rheumatoid arthritis (RA). Tumor necrosis alpha (TNF-α), an important mediator of inflammation in RA, regulates the circadian expression of clock proteins, and adenosine A(2A) receptors

Preladenant, a novel adenosine A(2A) receptor antagonist for the potential treatment of parkinsonism and other disorders.

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Adenosine A(2A) receptor antagonists exert antiparkinsonian effects in animal models and several drugs in this class are currently being assessed in clinical trials. Preladenant (SCH-420814) is an adenosine A(2A) receptor antagonist with a high affinity and very high selectivity for adenosine A(2A)

The effect of digoxin on contractility and fatigue of isolated guinea pig and rat hemidiaphragms.

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Digoxin could improve diaphragm contractility and fatigability if inhibition of sodium-potassium adenosine triphosphatase enhances calcium influx from extracellular sources, or it could impair contractility and worsen fatigue if it impairs maintenance of the membrane potential. We studied the

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