anisodamine/stroke

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A combination of neostigmine and anisodamine protects against ischemic stroke by activating α7nAChR.

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BACKGROUND Increasing endogenous acetylcholine by neostigmine decreased the ischemic cerebral injury. The off-target action on muscarinic receptor produced a variety of adverse effects and limited the clinical application on stroke. OBJECTIVE We combined neostigmine with anisodamine and investigated

Involvement of acetylcholine-α7nAChR in the protective effects of arterial baroreflex against ischemic stroke.

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OBJECTIVE Decreased baroreflex sensitivity is associated with poor outcome in many cardiovascular diseases including stroke, but the molecular mechanism underlying this relationship is unclear. This work was designed to test the hypothesis that acetylcholine (ACh) and α7 nicotinic ACh receptor

[Influence of anisodamine on heat-stress in rats].

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OBJECTIVE To study whether previously intravenous injection of anisodamine can prevent endotoxemia of heat stroke of rats. METHODS Experimental animals were randomly divided into two groups, their average artery pressure, heart rate, survival time, survival rate and rectal temperature were measured

Comparison of myocardial microcirculatory perfusion after catheter-administered intracoronary thrombolysis with anisodamine versus standard thrombus aspiration in patients with ST-elevation myocardial infarction.

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To evaluate efficacy, safety and feasibility of targeted intracoronary injection using pro-urokinase combined with anisodamine (TCA) versus thrombus aspiration (TA) in ST-elevation myocardial infarction (STEMI) patients with high thrombus loads.The best

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