atrial flutter/edema

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Amiodarone given by three routes to terminate fetal atrial flutter associated with severe hydrops.

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BACKGROUND We describe the concurrent administration of amiodarone using three different routes in order to provide: 1) rapid and adequate fetal loading without giving unduly high doses to the mother, and 2) a maintenance dose to the fetus without risking repeated invasive procedures. METHODS Rapid

Transplacental digoxin therapy for fetal atrial flutter with hydrops fetalis.

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BACKGROUND Without timely treatment, fetal atrial flutter (AF) could result in congestive heart failure, hydrops fetalis and even fetal demise. METHODS Prenatal echocardiography was used to confirm AF and assess fetal cardiac function with cardiovascular profile score. Transplacental digoxin therapy

Two cases of atrial flutter with fetal hydrops: successful fetal drug therapy.

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We describe two cases of fetal atrial flutter associated with severe fetal hydrops which were unresponsive to digoxin but were successfully treated with flecainide acetate. Two cases of fetal atrial flutter were identified in fetuses with severe fetal hydrops on 3rd trimester ultrasonogram(28 weeks'

Pulmonary edema after cardioversion for paroxysmal atrial flutter: left ventricular diastolic dysfunction induced by direct current shock.

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This report describes a patient with the pulmonary edema after cardioversion for paroxysmal atrial flutter without organic heart disease. A 68-year-old man was admitted to hospital for paroxysmal atrial flutter. Antiarrhythmic agents were not effective, and direct current cardioversion was performed

Successful treatment of atrial flutter by repeated intraperitoneal and intra-amniotic injections of amiodarone in a fetus with hydrops.

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OBJECTIVE We report a case of nonimmune hydrops fetalis caused by atrial flutter, which was successfully treated by intraperitoneal and intra-amniotic injections of amiodarone. METHODS A 27-year-old woman presented at 30 weeks of pregnancy with hydrops fetalis caused by a fetal atrial flutter. As

[Congenital atrial flutter with hydrops fetalis caused by tocolytic therapy (author's transl)].

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A 31-year-old healthy woman received Ritodrine (Pre-Par) from the 26th week of gestation because of twin pregnancy. Three weeks before birth continous intravenous medication with the same drug was commenced because of premature uterine contractions. Five days later a heart rate of over 200/minute

Perinatal treatment of refractory atrial flutter with hydrops fetalis: a case report.

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OBJECTIVE Highlight the management of a critically ill premature hydropic baby with refractory atrial flutter (AF) and successful outcome without neurologic sequel at 1 year of follow-up. METHODS A 23-year-old pregnant woman, G1PO, presented with fetal tachycardia at 32 weeks. RESULTS Ultrasound

Acute pulmonary edema after overdrive for atrial flutter.

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Congenital atrial flutter and cardiac failure presenting as hydrops foetalis at birth.

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[Pulmonary edema following tachysystolia caused by atrial flutter in a subject with recent myocardial infarct: failure of medical therapy. Dramatic resolution by electric counter shock].

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[Successful treatment of fetal atrial flutter and hydrops by maternal administration of oral digoxin: a case report].

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Acute Pulmonary Edema: A Rare Cause of Dyspnea After Electrical Cardioversion.

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Direct current cardioversion is a common management option for termination of tachydysrhythmias, including atrial fibrillation and atrial flutter. It is generally safe and effective with infrequent reporting of side effects. Pulmonary edema is a rare complication with reported

Acute pulmonary edema associated with direct current cardioversion in a structurally normal heart.

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The transthoracic application of synchronized direct current cardioversion (DCC) is widely used to terminate atrial fibrillation, atrial flutter, and other supraventricular tachyarrhythmia. DCC is a highly effective method for acute restoration of sinus rhythm. Although DCC is a relatively safe and

Circus movement atrial flutter in the canine sterile pericarditis model. Activation patterns during initiation, termination, and sustained reentry in vivo.

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The mechanisms of single-loop reentry in a syncytium without anatomically predetermined pathways have not been shown. Using a "jacket electrode" with 111 bipolar electrodes in a nylon matrix, we mapped in situ the atrial epicardial surface during atrial flutter in dogs with sterile pericarditis. Of

Neurological outcome of children who were treated for fetal tachycardia complicated by hydrops.

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OBJECTIVE Fetal tachycardia is a condition associated with congestive heart failure and development of fetal hydrops, which may result in neurological morbidity and mortality. The aim of this study was to investigate the long-term outcome of hydropic fetuses. METHODS This was a retrospective study

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