atropine/hemorrhage

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Atropine pharmacokinetics are affected by moderate hemorrhage and hypothyroidism.

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Atropine is used both to treat a variety of clinical disorders and as an antidote to cholinesterase poisoning. While various conditions affect the physiologic responses to atropine, little is known of the pharmacokinetics of this drug except under resting conditions. Pharmacokinetic studies were

Prevention of acrylonitrile-induced gastrointestinal bleeding by sulfhydryl compounds, atropine and cimetidine.

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We have recently demonstrated that acrylonitrile (VCN) causes acute gastric hemorrhage and mucosal erosions. The current studies were undertaken to investigate the effects of the sulfhydryl-containing compounds, cysteine and cysteamine, the cholinergic blocking agent atropine and the histamine H2

Effects of a dexmedetomidine constant rate infusion and atropine on changes in global perfusion variables induced by hemorrhage followed by volume replacement in isoflurane-anesthetized dogs.

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OBJECTIVE To evaluate the effects of a dexmedetomidine constant rate infusion (CRI) and atropine on changes in global perfusion variables induced by hemorrhage and volume replacement (VR) in isoflurane-anesthetized dogs. METHODS 8 adult dogs. METHODS Each dog was anesthetized twice, with a 2-week

EFFECT OF ATROPINE ON FETAL BRADYCARDIA AND ARTERIAL OXYGENATION: EXPERIMENTAL STUDY IN THE DOG DURING GRADED HEMORRHAGE AND FOLLOWING VASOPRESSOR ADMINISTRATION.

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[Atropine therapy of hemorrhagic menstruation disorders].

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Atropine influence on altered hemodynamics of the primate kidney in hemorrhagic shock.

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Effects of atropine, 2-PAM, or pyridostigmine in euvolemic or hemorrhagic conscious swine.

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[Experiences in the treatment of obstetrical uterine hemorrhages by the use of atropine].

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Stellate ganglion blockade and atropine do not affect the electrocardiographic changes of subarachnoid hemorrhage.

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Acute hemorrhage causes hepatic insulin sensitizing substance (HISS)-dependent insulin resistance.

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Hepatic insulin sensitizing substance (HISS) has been shown to account for 55% of the action of insulin in the fed state. HISS blockade leads to HISS-dependent insulin resistance (HDIR). The objective of this study was to test the hypothesis that insulin resistance produced by hemorrhage was HDIR.

Intracerebroventricular choline increases plasma vasopressin and augments plasma vasopressin response to osmotic stimulation and hemorrhage.

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Intracerebroventricular (i.c.v.) injection of choline (50-150 microg), a precursor of the neurotransmitter acetylcholine, produced a time-and dose-dependent increase in plasma vasopressin levels in conscious, freely moving rats. The increase in plasma vasopressin in response to i.c.v. choline (150

A correlation between multiple unit activity in the hypothalamus and electrocardiographic changes during a subarachnoid hemorrhage.

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A sudden onset and short latency of cardiovascular responses that may follow a subarachnoid hemorrhage (SAH) in the vicinity of the circle of Willis implicate neural mechanisms. To investigate this, multi unit activity (MUA) was recorded from the posterolateral hypothalamus and electrocardiogram was

Alteration in heart rate response to hemorrhage in conscious dogs with volume overload.

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The heart rate response to hemorrhage was studied in conscious dogs before and up to 2 mo after the establishment of volume overload due to systemic arteriovenous (a-v) fistulas. Before a-v fistula, heart rate increased markedly during hemorrhage. When hemorrhage was preceded by dextran infusion,

Venoconstriction of hepatic capacitance vessels during hemorrhage in cats: efferent mechanisms.

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Cats anesthetized with pentobarbital sodium were hemorrhaged (1 ml.min-1.kg body wt-1) until arterial pressure declined to 55 mmHg. Hepatic volume was recorded by plethysmography. In controls, 20 +/- 5 (SD) ml/kg was removed and hepatic volume decreased 3.6 +/- 0.8 ml/kg. Splanchnic nerve section or

Adrenocorticotropic hormone (ACTH) and centrally-acting cholinomimetic drugs improve survival of rats with severe hemorrhagic shock through distinct central cholinergic mechanisms.

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Pharmacological doses (40-160 micrograms/kg) of adrenocorticotropic hormone (ACTH) intravenously injected to urethane-anesthetized rats subjected to otherwise lethal hemorrhagic shock (mean arterial pressure stabilized at 20-25 mmHg) promptly restore blood pressure to about the pre-bleeding values,

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