blister/edema

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Acute edema blisters in a hereditary angioedema cutaneous attack.

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Hereditary angioedema is a rare autosomal dominant disease characterized by recurrent episodes of acute edema affecting the skin and the respiratory and digestive tracts. Acute edema blisters or hydro-static bullae develop after rapid accumulation of interstitial fluid usually associated to cardiac

Edema Bullae Mimicking Disseminated Herpes Zoster.

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Edema bullae typically forms at the site of skin swelling during acute states of volume overload, most commonly during renal or cardiac failure. Herpes zoster is a reactivation of latent varicella zoster virus that typically presents as painful vesicles in a dermatomal distribution. In

Blisters. Bullae secondary to edema.

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[Test of resorption time of intraderma edema bullae of artificial serum in obliterating arteritis of the extremities].

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Acute Edema Blisters on a Skin Swelling: An Unusual Manifestation of Hereditary Angioedema.

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[Test of the resorption time of intradermal edema bullae due to artificial serum in obliterating arteritis of the extremities].

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Case records of the Massachusetts General Hospital. Weekly clinicopathological exercises. Case 41-1989. A 65-year-old man with fever, bullae, erythema, and edema of the leg after wading in brackish water.

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[Surgically repaired bilateral giant bullae after long-time follow-up; report of a case].

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A 44-year-old male was admitted to our hospital owing to severe dyspnea of sudden onset. Chest X-ray disclosed bilateral giant bullae. His pulmonary function was severely depressed. VC (%) was 2.61 l (66.3%) and FEV1% (%) was 1.47 l (64.5%). Indication of surgical repair of giant bullae are; space

Re-expansion pulmonary edema following puncture of a giant bulla.

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Ipsilateral pulmonary edema may occur in a lung that has been rapidly reinflated after a period of collapse. The syndrome of re-expansion pulmonary edema is associated with variable degrees of hypotension and hypoxemia. In its extreme form, it may result in cardiac arrest and death. The initial

Localized bullous eruption in a patient with anasarca.

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The diagnosis of bullae can be a challenge. The proper diagnosis and prompt treatment is mandatory because most of these diseases are associated with a significant degree of morbidity. The authors present the case of a 76-year-old woman admitted for treatment of anasarca. She also exhibited a

Epistaxis and bullae in cynomolgus macaques (Macaca fascicularis).

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Epistaxis and bullae occurred in 35 of 54 cynomolgus macaques (Macaca fascicularis). Individual cases developed randomly during a 3 to 4 week period in the winter, and resolved within a week of onset. Clinical signs included nasal and eyelid swelling, bloody nasal discharge, sneezing, and bullous

Fracture blisters.

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Fracture blisters can pose a problem when dealing with trauma. The potential to increase the complication rate, delay surgery, and prolong wound healing are possible sequelae. Edema has to be controlled, however, because the blisters can form in spite of ice, elevation, and compression. They may

[Recurrent skin blisters for more than 7 months in a girl aged 15 months].

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A girl, aged 15 months, attended the hospital due to recurrent skin erythema, blisters, and desquamation for more than 7 months. Giemsa staining and immunohistochemical staining showed mast cell infiltration and degranulation. Hematoxylin staining showed spinous layer edema and blister formation

Histogenesis of UV light-induced blister formation.

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Histological examination of ultraviolet light (UVL)-induced blister formation in guinea pig skin is reported. Light and electron-microscopic examination was performed on skin exposed to single and multiple exposures of a large dose of UVB. In light microscopy, intracellular edema was prominent,

Herpes gestationis--bullae from hormones that cause increased extracellular matrix (autoantibodies to epidermal basement membrane as a secondary phenomenon).

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Herpes gestationis is a bullous skin disease clearly secondary to the hormones of pregnancy and other hormonal influences. It is the result multiple hormones. No one hormone is specific. The hormones increase ground substance viscosity in the skin and this induces edema and bullae formation. Only 10

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