cocaine/infarction

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Myocardial infarction with topical cocaine anesthesia for nasal surgery.

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Cocaine, the active alkaloid in coca leaf, is widely used as local anesthetic for otolaryngologic procedures. Our patient suffered an acute nontransmural myocardial infarction following clinical use of cocaine as topical anesthesia for nasal surgery, the first such case to be documented, to our

Acute myocardial infarction and chest pain syndromes after cocaine use.

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Seventy patients hospitalized with chest pain after cocaine use were retrospectively evaluated to define the risk and clinical course of acute myocardial infarction (AMI). AMI developed in 22 patients (31%) and transient myocardial ischemia was seen in an additional 9 patients (13%). Coronary risk

Cocaine-induced acute myocardial infarction.

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Acute myocardial infarction may occur following cocaine use. Cocaine-induced infarction is particularly common in younger patients aged 18 to 45 years old. Patients may or may not have angiographic evidence of coronary artery disease at the time of their acute event. Previous studies have shown that

Cocaine-associated myocardial infarction.

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Myocardial ischaemia and infarction has become a well-recognized sequelae of cocaine use. The possibility of recent cocaine use should be assessed in patients with potential myocardial ischaemia because the treatment of patients with myocardial ischaemia related to cocaine differs from that of

Carvedilol therapy after cocaine-induced myocardial infarction in patients with asthma.

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BACKGROUND Cocaine-induced myocardial infarction (MI) is well documented. Current literature recommends avoiding beta-blockers in the acute care setting, but after discharge from the hospital, benefits of beta-blocker use may outweigh risks in patients with recent MI resulting from cocaine use.

Acute myocardial infarction in two young male users of cocaine treated with thrombolytic therapy.

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Two young men with a history of cocaine addiction were admitted to our Coronary Unit because of acute myocardial infarction, and treated with thrombolytic agents. The involved coronary artery was found to be totally occluded in both of them. We suggest that spasm may constitute one of the primary

Cocaine Induced Bilateral Posterior Inferior Cerebellar Artery and Hippocampal Infarction.

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Cocaine is one of the most commonly abused recreational drugs, second only to marijuana. It blocks the reuptake of neurotransmitters such as norepinephrine and dopamine, that leads to persistent post-synaptic stimulation responsible for its excitatory effects. Cocaine-related strokes, both ischemic

[Renal and spleen infarction after massive consumption of cannabis and cocaine in a young man].

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Cannabis is the most widely consumed drug in the world, particularly among young subjects. Cocaine is the third leading illicit drug. Cases of renal infarction associated with combined consumption of cannabis and cocaine have been reported in the literature. We describe the case of a 24-year-old man

Acute and chronic cocaine exposure can produce myocardial ischemia and infarction in Yucatan swine.

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The purpose of this study was to determine whether the acute and chronic administration of cocaine could induce myocardial infarction. Twenty-five minipigs were studied before and 4 months after balloon angioplasty of the left anterior descending artery (LAD) and balloon denudation of the left

Cocaine-associated myocardial infarction. Clinical safety of thrombolytic therapy. Cocaine Associated Myocardial Infarction (CAMI) Study Group.

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OBJECTIVE To determine the safety of thrombolytic use in patients with cocaine-associated myocardial infarction. METHODS Retrospective cross-sectional survey. METHODS Twenty-nine acute care institutions. METHODS Patients who sustained cocaine-associated myocardial infarction from 1987 to 1993 were

Cocaine-induced transmural myocardial infarction in a Yorkshire swine with normal coronary arteries: Evidence for microvascular and/or epicardial coronary artery spasm.

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Cocaine-induced myocardial infarction with normal coronary arteries is well documented in humans. The exact mechanism of action remains speculative. We report one case of cocaine-induced myocardial infarction with normal coronaries in one swine. Systemic hemodynamics and angiographic,

High serum tryptase value in massive acute myocardial infarction with ventricular arrhythmia exortion in a cocaine abuser.

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A 38-year-old cocaine abuser was admitted to the Emergency Department with a one hour history of precordial chest pain associated with shortness of breath and extreme discomfort. On admission his blood pressure was 90/60 mmHg, the resting 12-lead ECG showed ventricular tachycardia at about 300 beats

Cocaine-induced renal infarction: report of a case and review of the literature.

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BACKGROUND Cocaine abuse has been known to have detrimental effects on the cardiovascular system. Its toxicity has been associated with myocardial ischemia, cerebrovascular accidents and mesenteric ischemia. The pathophysiology of cocaine-related renal injury is multifactorial and involves renal

Cerebral infarction associated with cocaine use.

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We report the case of a young man with an acute infarction of the left putamen and caudate nucleus, whose symptoms appeared six hours after intranasal use of approximately 0.5 g of cocaine hydrochloride. It seems probably that in this patient cocaine consumption played a role in the development of

[Renal infarction and acute renal failure induced by cocaine].

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We describe the case of a 36 year old man, habitual consumer of cocaine, who after the inhaled cocaine consumption develops acute renal failure secondary to massive left and segmental right renal infarction. Although the most frequent complications associated to cocaine consumption are of

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