corneal neovascularization/edema

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Penetrating Keratoplasty for Keratoconus With and Without Resolved Corneal Hydrops: Long-term Results.

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To evaluate the long-term risk of endothelial rejection, graft survival, and associated factors following penetrating keratoplasty (PK) for keratoconus, with and without prior resolved corneal hydrops. Retrospective cohort study. Primary outcome measures were endothelial rejection-free survival and

Comparison of the inhibitory effect of different doses of subconjunctival bevacizumab application in an experimental model of corneal neovascularization.

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OBJECTIVE To evaluate the inhibitory effect of subconjunctival bevacizumab as single- and multiple-dose application, and compare their effects on corneal neovascularization in a rat model. METHODS Thirty adult Sprague-Dawley rats were used in this experimental study. The central cornea of the rats

[Mechanisms of corneal neovascularization and modern options for its suppression].

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Quite a number of pathological factors exist that can disturb the balance between anti-angiogenic and proangiogenic mechanisms, thus causing vascularization of the cornea. The neovessels are immature, ill-formed, and show increased permeability, which is dangerous of corneal edema, lipid deposition,

Comparison of the effects of bevacizumab and ranibizumab injection on corneal angiogenesis in an alkali burn induced model.

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OBJECTIVE To investigate the effects of bevacizumab and ranibizumab on corneal neovascularization in an alkali burn-induced model of corneal angiogenesis. METHODS Fifteen Wistar albino rats were divided randomly into 3 groups after chemical cauterization of the cornea. The first group received a

Topical treatment of corneal alkali burns with Gly-thymosin β4 solutions and in situ hydrogels via inhibiting corneal neovascularization and improving corneal epidermal recovery in experimental rabbits.

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OBJECTIVE Corneal alkali burns are a severe disease and commonly encountered in the emergent clinic. A rapid medical treatment for the burn is very important. Gly-thymosin β4 (Gly-Tβ4) is a biomimic derivative of natural thymosin β4. The aim of this study is to evaluate the corneal recovery effects

Experimental inhibition of corneal neovascularization by endostatin gene transfection in vivo.

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OBJECTIVE To investigate endostatin gene therapy of rat corneal neovascularization induced by acid cauterization. METHODS pBlast-hEndostatin and pBlast-Mcs were identified by digestion with Nhe Iand Sal I, by PCR reaction, by sequence, and then by alignment of PCR products with the gene Bank using

Treatment of corneal neovascularization in ocular chemical injury with an off-label use of subconjunctival bevacizumab: a case report.

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BACKGROUND In this report, we describe the case of a patient with ocular chemical injury, symblepharon, and corneal neovascularization in whom subconjunctival injection of bevacizumab caused regression of corneal opacification and neovascularization, which led to visual improvement. METHODS A

Inhibitory effects of 90Sr/90Y β-irradiation on alkali burn-induced corneal neovascularization in rats.

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The aim of the present study was to investigate the inhibitory effects of 90Sr-90Y β-irradiation in a rat model of alkali burn-induced corneal neovascularization (CNV). Alkali burn-induced CNV was induced in the right eyes of 30 female Wistar rats, which were randomly divided into the following

The inhibitory effect of different concentrations of KH902 eye drops on corneal neovascularization induced by alkali burn.

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OBJECTIVE The aim of this study was to evaluate the inhibitory effect of different concentrations of KH902 eye drops on rabbit corneal neovascularization (CNV) induced by alkali burn. METHODS Forty-eight adult rabbits were randomized into four groups after alkali burning: Group A (2.5 mg/ml), Group

[Inhibition of corneal neovascularization by vascular endothelial growth inhibitor gene].

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OBJECTIVE To evaluate the effect of Effectene lipofectene mediated plasmids encoding human pcDNA4-vascular endothelia growth inhibitor (pcDNA4-VEGI) gene on corneal neovascularization (CNV). METHODS It was an experimental study. Forty healthy New Zealand albino rabbits (40 eyes) were divided into 4

[Morphological study of experimental corneal neovascularization after anterior uveal ischemia].

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We morphologically investigated what kinds of inflammatory cells infiltrate the corneoscleral limbus by light and electron microscopy and what manner of keratocytes and vascular endothelial cells appear at the corneal limbus, when the corneal edema was aggravated severely and clinical corneal new

Sustained-release endotoxin. A model for inducing corneal neovascularization.

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The rabbit corneal pocket assay is one of the most frequently used systems for the study of angiogenesis. This model particularly is useful to identify stimulators of new blood vessel formation. More recently, however, interest in inhibitors of angiogenesis has grown, and several antiangiogenic

Topical formulations of novel angiostatic steroids inhibit rabbit corneal neovascularization.

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OBJECTIVE To evaluate the antiangiogenic potential of topical ophthalmic formulations of the novel angiostatic steroids AL-3789 and AL-4940, using a rabbit model of corneal neovascularization. METHODS Neovascularization was induced in the rabbit cornea by surgical implantation of a standard ethylene

Topical and subconjunctival ranibizumab (lucentis) for corneal neovascularization in experimental rat model.

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OBJECTIVE To compare the effectiveness of the topical and subconjunctival (SC) ranibizumab treatment in experimental corneal neovascularization (NV) model in rats. METHODS A model of NV was generated by cauterizing right corneas of 30 Sprague-Dawley rats with silver nitrate. The animals were

[Animal experiment studies on the role of inflammation mediators in corneal neovascularization].

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Natural and synthetic inflammatory compounds were implanted in the corneas of rabbits to clarify the question whether corneal neovascularization is induced by stromal edema alone, or by neovascular mediators. It could be demonstrated that prostaglandin E1 and E2 have an angiogenetic capacity,

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