coronary occlusion/edema

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Computerized tomography of ischemic myocardium: quantitation of extent and severity of edema in an in vitro canine model.

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The capacity of computerized tomography to assess myocardial edema resulting from acute coronary occlusion was investigated in 19 arrested and 2 beating canine hearts. Edematous myocardium was consistently detected as areas of decreased attenuation values. The magnitude of the decrease in

The effect of ethanol on arrhythmias and myocardial necrosis in rats with coronary occlusion and reperfusion.

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Ethanol (1, 2 and 3 g/kg, intravenously) decreased the severity of the ischemic arrhythmias in rats with ligation of the left coronary artery and subsequent coronary reperfusion. Reperfusion arrhythmias occurring intensively after occlusion times of 5 and 15 min, respectively, were however not

Effect of reduction in myocardial edema on myocardial blood flow and ventricular function after coronary reperfusion.

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The contribution of myocardial edema to the no-reflow phenomenon, left ventricular functional recovery, and infarct size after coronary occlusion and reperfusion is uncertain. To examine this, we studied 26 open-chest dogs after coronary occlusion and reperfusion. Twelve dogs received hypertonic

Regional myocardial blood flow, edema formation, and magnetic relaxation times during acute myocardial ischemia in the canine.

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This study was designed to measure early changes in myocardial perfusion after acute coronary occlusion, and to examine the relationships among blood flow, myocardial edema, and magnetic relaxation times of ex vivo myocardial tissue. In ten dogs, the left anterior descending coronary artery was

Quantitative tracking of edema, hemorrhage, and microvascular obstruction in subacute myocardial infarction in a porcine model by MRI.

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Pathophysiological responses after acute myocardial infarction include edema, hemorrhage, and microvascular obstruction along with cellular damage. The in vivo evolution of these processes simultaneously throughout infarct healing has not been well characterized. The purpose of our study was to

Dissociation between anti-infarct effect and anti-edema effect of ischemic preconditioning.

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This study tested the hypothesis that preconditioning, by reducing catabolite accumulation during ischemia, reduces osmotic swelling and myocardial necrosis during subsequent reperfusion. Farm pigs were randomly allocated to one of three groups of treatment: a control group undergoing a 48-min

Pulmonary edema formation after myocardial infarction and coronary reperfusion: intravascular and extravascular pulmonary fluid volumes. IV.

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Two studies were performed to assess the importance of coronary reperfusion on pulmonary capillary endothelial permeability in 34 anesthetized dogs. The response of intravascular pulmonary blood volume (PBV) and extravascular lung water (EVLW) was examined using indicator-dilution and postmortem

Tissue edema and loss of tracer microspheres in infarcted myocardium.

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Microsphere loss from the ischemic myocardium was studied in a canine model after 2 and 7 days coronary occlusion. 10 million tracer microspheres (TM) of 7-10 micrograms diameter were injected into the left atrium before and 15 minutes after left anterior descending coronary artery (LAD) occlusion.

Edema as a very early marker for acute myocardial ischemia: a cardiovascular magnetic resonance study.

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OBJECTIVE This study was designed to determine whether imaging myocardial edema would identify acute myocardial ischemia before irreversible injury takes place. BACKGROUND Early identification of acute myocardial ischemia is a diagnostic challenge. METHODS We studied 15 dogs with serial

Pathophysiology Underlying the Bimodal Edema Phenomenon After Myocardial Ischemia/Reperfusion.

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BACKGROUND Post-ischemia/reperfusion (I/R) myocardial edema was recently shown to follow a consistent bimodal pattern: an initial wave of edema appears on reperfusion and dissipates at 24 h, followed by a deferred wave that initiates days after infarction, peaking at 1 week. OBJECTIVE This study

Myocardial edema as detected by pre-contrast T1 and T2 CMR delineates area at risk associated with acute myocardial infarction.

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OBJECTIVE The aim of this study was to determine whether cardiac magnetic resonance (CMR) in vivo T1 mapping can measure myocardial area at risk (AAR) compared with microspheres or T2 mapping CMR. BACKGROUND If T2-weighted CMR is abnormal in the AAR due to edema related to myocardial ischemia, then

Reperfusion conditions: importance of ensuring gentle versus sudden reperfusion during relief of coronary occlusion.

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This study tests the hypothesis that more muscle salvage after acute ischemia is possible by "gentle," temporary reperfusion than with sudden, complete revascularization. Ten dogs underwent 4 hours of left anterior descending coronary artery ligation with reperfusion during total vented bypass for 1

Pulmonary edema formation with myocardial infarction and left atrial hypertension: intravascular and extravascular pulmonary fluid volumes.

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The response of pulmonary blood volume (PBV) and extravascular lung water (EVLW) was examined by indicator-dilution techniques in 14 "open chest" dogs, seven that underwent coronary occlusion (group 2) and seven that served as controls (group 1). Data were obtained in a control stage (control stage

T₂ -weighted MRI of post-infarct myocardial edema in mice.

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T(2) -weighted, cardiac magnetic resonance imaging (T(2) w CMR) can be used to noninvasively detect and quantify the edematous region that corresponds to the area at risk (AAR) following myocardial infarction (MI). Previously, CMR has been used to examine structure and function in mice, expediting

Selective arterialization of coronary veins for diffuse coronary occlusion. An experimental evaluation.

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The selective arterialization of coronary veins as a surgical therapy for diffuse coronary arteriosclerosis was studied in canine and sheep experiments. The arterialized blood can be delivered promptly to the selected region of the myocardium and an adequate runoff exists for this mode of

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