coronary occlusion/nicotine

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Effects of cigarette smoke and nicotine on platelets and experimental coronary artery thrombosis.

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The causal link between smoking, atherosclerosis and an increased risk for acute platelet mediated coronary events such as acute platelet thrombus formation, myocardial infarction, and sudden coronary death is not clear. Our studies suggest that there may be a transient increase in in vivo platelet

[Acute myocardial infarct caused by nicotine-induced erythrocytosis].

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A 29-year-old heavy smoker presented with an acute myocardial infarction and hematocrit of 70%. At immediate coronary angiography a complete occlusion of the right coronary artery was found. After intracoronary urokinase the coronary arteries were found to be completely normal. Causes for the

Prostanoids and cardiac reflexes of sympathetic and vagal origin.

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Prostaglandins in concentrations too low to stimulate afferent nerve endings in the heart may sensitize them to chemical or mechanical stimuli that activate cardiac reflexes during myocardial ischemia. Bradykinin, which is released from the heart during ischemia, elicits sympathetically mediated

Lipid effects of smoking.

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Cigarette smoking is believed to cause harmful cardiovascular and atherogenic effects resulting from changes in lipid metabolism. Intravenous nicotine and smoking raise plasma free fatty acid (FFA) levels through enhanced lipolysis resulting from sympathoadrenal stimulation. The study reported here

Cardiac nociceptors and ischemia: role of sympathetic afferents in cat.

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In total, 86 units were recorded from T2 and T3 left thoracic rami of cat. These receptors were located on the circumflex coronary artery, anterior descending coronary artery, and its adjacent myocardial regions. The conduction velocity of these fibres was in the range of "C" (0.5 to 1.8 m/s) and "A

Rectal response of cardiac origin in the cat: involvement of nitric oxide and acetylcholine.

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Local application of nicotine over the surface of the left ventricle and also occlusion of the left anterior descending coronary artery in the lightly anaesthetised, open-chested, artificially ventilated cat resulted a biphasic rectal movement--initial relaxation followed by sustained contraction.

Adverse effects of cigarette smoke and induction of oxidative stress in cardiomyocytes and vascular endothelium.

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Active and passive exposure to cigarette smoke (CS) increases the risk of, and has deleterious effects in, ischaemic heart disease. Exposure to CS increases infarct size in experimental models of coronary occlusion and reperfusion. Among many possible mechanisms for these deleterious effects in

Effects of ischemia on cardiac afferent sympathetic and vagal reflexes in dog.

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To determine the time course of afferent sympathetic and vagal denervation after coronary occlusion and that of neural recovery after reperfusion, we measured the vasopressor responses to bradykinin and the vasodepressor responses to nicotine applied in a felt pad to the left ventricular epicardium

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