diabetic coma/seizures

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Neurologic manifestations of diabetic comas: correlation with biochemical alterations in the brain.

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Coma and other neurologic abnormalities are present in patients with either diabetic ketoacidosis (DKA) or nonketotic coma (NKC), and the cause of such phenomena are not known. Patients with NKC also manifest seizures and focal neurologic changes. Treatment of diabetic coma with insulin may induce

EDs grapple with record-breaking number of drug shortages.

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A record-breaking number of drug shortages is impacting all areas of health care, but ED and EMS operations are under added pressure to work around such shortages quickly to meet critical patient needs. Experts say the most successful organizations have established strong communication channels

Special considerations with the elderly client.

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The nationwide increase in the size of the elderly population is resulting in a significant increase in the number of speech--language and hearing clinicians who work with the elderly client. Certain considerations are required in working with this patient population, among the more important of

[Nontraumatic rhabdomyolysis: its etiology and the predictive factors of acute kidney failure].

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BACKGROUND The aim of this study was to determine the causes of non traumatic or medical rhabdomyolysis (RM) and analyze the clinical and biologic characteristics, as well as the predictive factors of acute renal failure (ARF). METHODS Fifty-nine patients with RM admitted to the Internal Medicine

Ion regulation in the brain: implications for pathophysiology.

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Ions in the brain are regulated independently from plasma levels by active transport across choroid plexus epithelium and cerebral capillary endothelium, assisted by astrocytes. In "resting" brain tissue, extracellular potassium ([K+]o) is lower and [H]o is higher (i.e., pHo is lower) than elsewhere

Rhabdomyolysis associated with hyperthyroidism.

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BACKGROUND Nontraumatic rhabdomyolysis has been associated with alcohol and drug abuse, seizures, strenuous exercise, muscle hypoperfusion, hyperthermia, electrolyte disturbances, diabetic coma, and hypothyroidism. Hyperthyroidism can be associated with several neuromuscular manifestations, such as

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