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diphenylhydantoin/edema
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7 results
Diphenylhydantoin and intermittent edema.
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Diphenylhydantoin-induced serum sickness with fibrin-platelet thrombi in lymph node microvasculature.
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In this 16 year old boy a syndrome, characterized by high fever, generalized lymphadenopathy, splenomegaly, diffuse skin rash, facial and periorbital edema, neutropenia, thrombocytopenia, elevated serum glutamic oxaloacetic transaminase (SGOT) levels and transient electrocardiographic changes,
Vasogenic brain edema in focal 4-aminopyridine seizures: the role of neuronal hyperactivity.
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The protein transport of the neocortical blood vessels during experimental focal seizures was investigated in adult Wistar rats. The seizure focus was produced by the local application of isosmotic and isohydric 4-aminopyridine solution, while the electrocorticogram was monitored. The protein
The role of dilantin in the prevention of pulmonary edema associated with cerebral hypoxia.
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Twenty-five mongrel dogs were subjected to isolated cerebral hypoxia or noncerebral systemic hypotension to create the congestive pulmonary lesions of the shock lung syndrome (SLS). Eleven of the experimental dogs were pretreated with 5, 5-diphenylhydantoin (DPH). Prophylactic DPH protects the lung
[Sodium diphenylhydantoin as a probable cause of pancreatitis].
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BACKGROUND
Drug-induced pancreatitis is much more common in children than in adults. Many drug have been implicated in its pathogenesis. Among the neuropsychiatric drugs only valproic acid, carbamazepine, clozapine and ergotamine have been reported. Phenytoin is commonly used for the treatment of
Behenoyl cytarabine-associated reversible encephalopathy in a patient with acute myelogenous leukemia.
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We report a case of reversible encephalopathy syndrome in a 16-year-old girl with acute myelogenous leukemia (AML), who is undergoing during consolidation chemotherapy composed of BH-AC (N4-behenoyl-1-beta-D-arabinofuranosyl cytosine) and idarubicin. On the 6th day of chemotherapy, she was in a
Altered Na(+)-channel function as an in vitro model of the ischemic penumbra: action of lubeluzole and other neuroprotective drugs.
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Veratridine blocks Na(+)-channel inactivation and causes a persistant Na(+)-influx. Exposure of hippocampal slices to 10 microM veratridine led to a failure of synaptic transmission, repetitive spreading depression (SD)-like depolarizations of increasing duration, loss of Ca(+)-homeostasis, a large
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