diphenylhydantoin/hypoxia

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Diphenylhydantoin attenuates hypoxia-induced release of [3H]glutamate from rat hippocampal slices.

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The ability of diphenylhydantoin (DPH) to prevent hypoxia-induced [3H]glutamate release was examined in perfused rat hippocampal slices. Hypoxia (25 min; 95% N2/5% CO2) caused a prolonged release of [3H]glutamate, which was reduced significantly if DPH (20 microM) was present from the beginning of

Diphenylhydantoin protects against hypoxia-induced impairment of hippocampal synaptic transmission.

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The ability of diphenylhydantoin (DPH) to protect against hypoxia-induced neuronal damage was examined using electrophysiological recordings of extracellular evoked potentials from CA1 pyramidal neurons of rat hippocampal slices in vitro. In normal medium, a 15-min hypoxic insult (95% N2/5% CO2)

The effect of diphenylhydantoin on the electroretinogram of rabbits. II. Effects of hypoxia and potassium.

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"Protective" action of diphenylhydantoin on canine Purkinje fibers during hypoxia.

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EFFECTS OF DIPHENYLHYDANTOIN ON THE DURATION OF RESPIRATORY ACTIVITY DURING ANOXIA.

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Sudden death following intravenous sodium diphenylhydantoin.

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A well-documented (by ECG as well as autopsy) case of fatal ventricular asystole secondary to intravenous administration of sodium diphenylhydantoin is presented. The possible role of ventricular conduction disturbances, hypoxemia, and metabolic abnormalities as precipitating factors are discussed.

The role of dilantin in the prevention of pulmonary edema associated with cerebral hypoxia.

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Twenty-five mongrel dogs were subjected to isolated cerebral hypoxia or noncerebral systemic hypotension to create the congestive pulmonary lesions of the shock lung syndrome (SLS). Eleven of the experimental dogs were pretreated with 5, 5-diphenylhydantoin (DPH). Prophylactic DPH protects the lung

Effects of non-opioid antitussives on hypoxia-induced electrical changes in rat hippocampal slices: a comparative study with anticonvulsant drugs.

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1. The effects of the non-opioid antitussives caramiphen and carbetapentane and of the anticonvulsants 5,5-diphenylhydantoin and MK 801 were tested towards hypoxia-induced electrical changes in rat hippocampal slices. 2. The incidence of appearance of hypoxia-induced epileptiform bursting was

Phenytoin protects against hypoxia-induced death of cultured hippocampal neurons.

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The neuroprotective actions of the anticonvulsant phenytoin (diphenylhydantoin, PHT) were evaluated using 3 week old primary hippocampal cultures derived from 19 day embryonic rat. When added to the culture medium prior to a hypoxic insult, PHT increased neuronal viability two-fold. Doubling

Enhanced sensitivity of hippocampal pyramidal neurons from mdx mice to hypoxia-induced loss of synaptic transmission.

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The gene at the Duchenne/Becker muscular dystrophy locus encodes dystrophin, a member of a protein superfamily that links the actin cytoskeleton to transmembrane plasmalemmal proteins. In mature skeletal myocytes, the absence of dystrophin is associated with decreased membrane stability, altered

Hypothermia as a mechanism for drug-induced resistance to hypoxia.

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A linear regression line relating the logarithm of survival time in a standard hypoxia survival test (4.6% oxygen) with hypothermia was obtained upon cooling mice by exposure to either a graded hypoxia or a cold environment. When S/BT values (log minutes survival versus body temperature) of

[Treatment of status epilepticus in the adult. Retrospective analysis of 192 cases treated in intensive care units].

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The results of therapy have been analyzed in a series of 192 patients admitted for status epilepticus over 7 years in two intensive care units. Most (142 cases without any prior epilepsy) corresponded to secondary forms. In 2/3 of the cases, the patients were admitted because of failure of

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