estradiol 17 beta/inflammation

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Effect of mimicking prepartum concentration of estradiol-17 beta on the inflammatory response to endotoxin in gilts.

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The effect of mimicking prepartum concentration of estradiol-17 beta on the inflammatory response to endotoxin in gilts was studied. The study was performed in a split-litter design and comprised 5 pairs of littermates. A catheter was inserted into the jugular vein 2 days prior to the start of the

[Estradiol-17 beta and progesterone levels in the blood of patients with infertility of inflammatory origin].

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Regulation of alpha 1-acid glycoprotein plasma concentration by sex steroids and adrenal-cortical hormones during experimental inflammation in the rat.

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Treatment of adult intact rats with sex steroids (estradiol-17 beta, ethynylestradiol, dihydrotestosterone) raises the concentration of serum acute-phase alpha 1-acid glycoprotein (AGP). Estrogens are more effective than dexamethasone, and experimental inflammation causes an additive effect on AGP

Female steroid hormones regulate production of pro-inflammatory molecules in uterine leukocytes.

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Estrogens and progesterone could be among the environmental signals that govern uterine immune cell synthesis of pro-inflammatory substances. In order to investigate this possibility, we first mapped expression of the inducible nitric oxide synthase (iNOS) and tumor necrosis factor-alpha (TNF-alpha)

Solidagenone from Solidago chilensis Meyen inhibits skin inflammation in experimental models

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Solidagenone (SOL) is a labdane-type diterpenoid found in Solidago chilensis, a plant traditionally used to treat skin diseases, kidney pain and ovarian inflammation. In this study, the topical anti-inflammatory activity of SOL was evaluated using in vivo and in silico assays. Croton-oil-,

Activation and distribution of inflammatory cells in the mouse uterus during the preimplantation period.

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Northern blot analysis of mouse uterine RNA showed that IL-1 (alpha and beta), and TNF-alpha mRNA were abundant on day (D) 1 of pregnancy, reduced on D2, and remained basal throughout the remainder of the preimplantation period (D3 and D4). Elevated IL-1 beta and TNF-alpha mRNA levels on D1 were

Sex-hormone-induced inflammation and fibromuscular proliferation in the rat lateral prostate.

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The subacute administration of estradiol-17 beta was shown to be a potent inducer of an inflammatory response specific to the lateral prostate of the castrated Wistar rat. The subsequent administration of dihydrotestosterone restored the wet weight of the gland while maintaining the inflammation

Cytokine-induced production of IFN-beta 2/IL-6 by freshly explanted human endometrial stromal cells. Modulation by estradiol-17 beta.

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The cytokine IFN-beta 2/IL-6 has emerged as an important means of communication between cells--both within the immune system as well as outside it. In exploring the link between the endocrine and the immune systems, we have studied the secretion of IFN-beta 2/IL-6 by freshly explanted human

The effects of 17 alpha-estradiol to inhibit inflammation in vitro.

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17 Alpha-estradiol (17 α-E2) is a natural, non-feminizing stereoisomer of 17 beta-estradiol (17 β-E2). Whereas much is known about the physiological effects of 17 β-E2, much less is known about 17 α-E2. For example, 17 β-E2 exerts anti-inflammatory effects in neurons and adipocytes through binding

Changes in serum concentration and mRNA level of rat C-reactive protein.

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C-reactive protein (CRP) is a typical acute-phase protein found in many animals. Several stimuli, including IL-6 produced by macrophages localized around the site of tissue injury and/or bacterial infection, induce CRP synthesis in liver. One of the authors (W.N.) reported previously that sex

Sulindac and its metabolites inhibit multiple transport proteins in rat and human hepatocytes.

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Sulindac is a commonly used nonsteroidal anti-inflammatory drug. This study tested the hypothesis that sulindac-mediated drug-drug interactions and/or hepatotoxicity may be caused, in part, by inhibition of proteins responsible for the hepatic transport of drugs and/or bile acids by sulindac and/or

Matrix metalloproteinases in the pathogenesis of estradiol-induced nonbacterial prostatitis in the lateral prostate lobe of the Wistar rat.

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Chronic nonbacterial prostatitis develops spontaneously with age in the lateral lobe of the prostate in some strains of rat. Our objective was to examine the role of matrix metalloproteinases (MMP) in the pathogenesis of chronic nonbacterial prostatitis using a chronic estrogen treatment, Wistar rat

Plasma protein and hormone profiles associated with autoimmune oophoritis and ovarian tumorigenesis in neonatally thymectomized mice.

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Interactions between the immune and endocrine systems may have an important role in ovarian tumorigenesis. Neonatal thymectomy at 3 days of age (Tx-3) in (C3H/HeMs x 129/J)F1 (C31) female mice results in an autoimmune ovarian dysgenesis then subsequent tumor formation. At 3 months of age the

Antigens in bovine estrual and cystic follicles.

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The antigenic spectra of estrual or cystic follicular fluid (FF) consisted of 11 blood serum antigens while that of estrual or cystic ovary consisted of 9 identical antigens of which 2 were ovarian specific. The two ovarian-specific antigens were detected with antisera to estrual or cystic ovary but

Aspiration cytology and E2 content in ovarian tumors.

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This study demonstrates the reasonable reliability of fine needle aspiration cytology in ovarian tumors: 91% of benign and 84.5% of malignant tumors were diagnosed correctly by aspiration cytology. The concentration of estradiol-17 beta in cyst fluid may be helpful in distinguishing follicle cysts

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