gluten/dental caries

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[Effect of gluten-dependent coeliac disease on the condition of oral cavity and teeth in children].

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After a review of the pertinent literature the effect is described of gluten-dependent coeliac disease on the condition of the oral cavity and teeth in children. Attention is called to the condition of the parodontium on the occurrence of developmental abnormalities of hard dental tissuses and

Discovery of a novel and rich source of gluten-degrading microbial enzymes in the oral cavity.

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BACKGROUND Celiac disease is a T cell mediated-inflammatory enteropathy caused by the ingestion of gluten in genetically predisposed individuals carrying HLA-DQ2 or HLA-DQ8. The immunogenic gliadin epitopes, containing multiple glutamine and proline residues, are largely resistant to degradation by

Nutrition and caries. III. The reproducibility of the caries scoring methods for rats and the influence of gluten on experimental rat caries.

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[The influence of a gluten diet and its components on nutrition and the development of dental caries in rats].

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Oral health status and salivary properties in relation to gluten-free diet in children with celiac disease.

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BACKGROUND Patients with celiac disease (CD) have a wide variety of symptoms, from being asymptomatic to having chronic diarrhea, abdominal pain, and extraintestinal symptoms. In the oral cavity, enamel defects and recurrent aphthous stomatitis are the most common symptoms. The aim of the study was

Celiac and Non-Celiac Forms of Gluten Sensitivity: Shifting Paradigms of an Old Disease.

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Gluten sensitivity is one of the prominent features of celiac disease (CD) which is an autoimmune disorder characterized by damaged lining of the small intestine. CD was known already to ancient Greeks as κοιλιακός (keeleeakoss) i.e. disease of the abdominal cavity hence celiac. Focus of this

Oral mucosal changes in coeliac patients on a gluten-free diet.

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Oral mucosal lesions or dental enamel defects may be the only presenting features of coeliac disease. A series of 128 patients with coeliac disease (CD) on a gluten-free diet (GFD), 8 patients with a newly diagnosed CD, and 30 healthy controls participated in a clinical and histopathological study

[Analysis of the leading pathogenetic factors of dental caries in children with celiac disease]

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The aim: To identify the leading pathogenetic factors of dental caries in children with celiac disease. Material and methods: The study included 48 children with a confirmed

Effect of Rothia mucilaginosa enzymes on gliadin (gluten) structure, deamidation, and immunogenic epitopes relevant to celiac disease.

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Rothia mucilaginosa, a natural microbial inhabitant of the oral cavity, cleaves gluten (gliadin) proteins at regions that are resistant to degradation by mammalian enzymes. The aim of this study was to investigate to what extent the R. mucilaginosa cell-associated enzymes abolish gliadin immunogenic

Selective PQQPFPQQ Gluten Epitope Chemical Sensor with a Molecularly Imprinted Polymer Recognition Unit and an Extended-Gate Field-Effect Transistor Transduction Unit.

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A molecularly imprinted polymer (MIP) recognition system was devised for selective determination of an immunogenic gluten octamer epitope, PQQPFPQQ. For that, a thin MIP film was devised, guided by density functional theory calculations, and then synthesized to become the chemosensor recognition

Pharmaceutically modified subtilisins withstand acidic conditions and effectively degrade gluten in vivo.

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Detoxification of gluten immunogenic epitopes is a promising strategy for the treatment of celiac disease. Our previous studies have shown that these epitopes can be degraded in vitro by subtilisin enzymes derived from Rothia mucilaginosa, a natural microbial colonizer of the oral cavity. The

Salivary Gluten Degradation and Oral Microbial Profiles in Healthy Individuals and Celiac Disease Patients.

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Celiac disease (CD) is a chronic immune-mediated enteropathy induced by dietary gluten in genetically predisposed individuals. Saliva harbors the second highest bacterial load of the gastrointestinal (GI) tract after the colon. We hypothesized that enzymes produced by oral bacteria may be involved

Changes in jejunal mucosa after long-term feeding of germfree rats with gluten.

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BACKGROUND Coeliac disease (CD) or gluten-sensitive enteropathy is a chronic gastrointestinal disease of children and adults. An experimental model using inbred germfree rats has been developed to study the effects of intragastric gliadin application on intestinal mucosa. METHODS AVN strain Wistar

[The condition of the stomatognathic system in children with gluten-dependent coeliac disease].

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Stomatological examination was carried out of 38 children with coeliac disease. The condition of teeth with regard to caries was better than in children without coeliac disease. Occlusal abnormalities were noted in one-third of patients, in one-third of children with mixed teeth the dental age was

Orally based diagnosis of celiac disease: current perspectives.

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Celiac disease (CD) is a lifelong immune-mediated disorder caused by the ingestion of wheat gluten in genetically susceptible persons. Most cases of CD are atypical and remain undiagnosed, which exposes the individuals to the risk of life-threatening complications. Serologic endomysial and tissue

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