gluten/stroke

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Preparation of corn peptide from corn gluten meal and its administration effect on alcohol metabolism in stroke-prone spontaneously hypertensive rats.

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Corn peptide (CP) was prepared from corn gluten meal by proteolysis with alkaline protease from alkalophilic Bacillus A-7. Free amino acids were not found in the CP product. Gel filtration on a Shodex OH-packed column revealed that the molecular weight distribution of the CP was less than about

Homocysteine and related B-vitamin status in coeliac disease: Effects of gluten exclusion and histological recovery.

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OBJECTIVE Hyperhomocysteinaemia is considered to be a risk factor for cardiovascular disease (particularly stroke) and has been implicated in recurrent miscarriage and osteoporotic fracture, recognized manifestations of coeliac disease (CD). The objective of this study was to compare plasma

Recurrent ischemic strokes in a young celiac woman with MTHFR gene mutation.

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Celiac disease (CD) is frequently associated with neurological disorders, but very few reports concern the association with ischemic stroke. A 26-year-old woman affected by CD with secondary amenorrhea, carrier of a homozygous 5,10-methylenetetrahydrofolate reductase mutation with

[Celiac disease and ischemic stroke].

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BACKGROUND Neurological manifestations of celiac disease are various. An association with ischemic stroke is not common and has not been well documented. We report two cases. METHODS The first patient had experienced several transient ischemic strokes in the past 2 years and then had an acute

Liver Transplantation for Fulminant Hepatic Failure Due to Heat Stroke: A Case Report.

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Heat stroke is a condition caused by an excessive increase in body temperature in a relatively short period of time, and is clinically characterized by central nervous system dysfunction, including delirium, seizures, coma, and severe hyperthermia. In this context, the resulting fulminant hepatic

Does cryptic gluten sensitivity play a part in neurological illness?

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BACKGROUND Antigliadin antibodies are a marker of untreated coeliac disease but can also be found in individuals with normal small-bowel mucosa. Because neurological dysfunction is a known complication of coeliac disease we have investigated the frequency of antigliadin antibodies, as a measure of

The choroid plexus and system disease in mental illness. III. The exogenous peptide hypothesis of mental illness.

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Based on the apparent existence of a second (choroid plexial) blood-brain barrier offering a new brain attack mechanism on the periventricular primary personality brain (Rudin, 1980) and which may be breached to produce the schizophreniform psychosis characteristic of systemic lupus erythematosus

[Neurologic complications in inflammatory bowel diseases].

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Some inflammatory diseases of the gastrointestinal system are associated with neurological symptoms which, in rare cases, may precede the onset of the gastrointestinal manifestation of the disease. Celiac sprue is characterized by an intolerance to the wheat protein gluten. The typical neurological

Cholesterol profile in people with newly diagnosed coeliac disease: a comparison with the general population and changes following treatment.

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Recent studies have suggested that untreated coeliac disease is associated with lower total cholesterol than in the general population while the effect of treatment with a gluten-free diet on the cholesterol profile of clinically apparent coeliac disease is not known. We measured the cholesterol

[Hypertransaminasaemia and impaired liver function in a patient with oligosymptomatic celiac disease].

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Hypertransaminasaemia and impaired liver function in a patient with oligosymptomatic celiac disease. We describe the case of a 45-year-old man who was referred for evaluation of elevated aminotransferases. One year before referral the patient developed an ischemic stroke followed by a subdural

Onset of Thrombin Generation Occurs More Rapidly in Pediatric Patients With Celiac Disease.

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OBJECTIVE Celiac disease (CD) is a risk factor for venous thromboembolism (VTE) and stroke, but the mechanisms are unclear. Continuous measurement of thrombin generation in plasma is a feasible way to detect hypercoagulable changes. The aim of this pilot study was to investigate thrombin generation

Wallenberg syndrome as a sole presentation of celiac disease.

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Celiac sprue is a gluten sensitive enteropathy in which there is a small bowel villous atrophy associated with gastrointestinal and extraintestinal symptoms. True prevalence is difficult to ascertain because many patients have atypical symptoms or none at all. Few children display CNS symptoms such

Extraintestinal considerations in inflammatory bowel disease.

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If one reviews the literature with zeal, it is increasingly apparent that few organs escape recruitment when IBD is chronic or progressive. Insights into mucosal pathophysiology have helped with understanding the more frequent extraintestinal manifestations, but the mechanisms attendant to the

Cardiovascular involvement in celiac disease.

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Celiac disease (CD) is an autoimmune response to ingestion of gluten protein, which is found in wheat, rye, and barley grains, and results in both small intestinal manifestations, including villous atrophy, as well as systemic manifestations. The main treatment for the disease is a gluten-free diet

[Intracardiac thrombosis during celiac disease].

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Thrombotic events occurring in the course of celiac disease are frequently reported in the literature. The localization is often unusual, mainly affecting the hepatic veins. To our knowledge, this is the first report of intracardiac thrombosis occurring in a patient with celiac disease. A

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