hydrothorax/albumin

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Paracetamol and ibuprofen block hydrothorax absorption in mice.

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OBJECTIVE Non-steroidal anti-inflammatory agents (NSAIDs) and paracetamol alter pleural permeability, hindering pleural fluid recycling. The aim of this study was to investigate the effect of different analgesic and anti-inflammatory agents on fluid recycling in an induced hydrothorax model in

[Refractory hydrothorax in primary biliary cirrhosis: successful treatment with transjugular intrahepatic portosystemic stent shunt].

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METHODS A 55-year-old woman with known primary biliary cirrhosis (PBC) was hospitalized because of increasing dyspnoea. A year before she had for the first time experienced a right-sided pleural effusion which had to be drained every 4 weeks. Physical examination revealed dullness on percussion and

Pleural fluid analysis and radiographic, sonographic, and echocardiographic characteristics of hepatic hydrothorax.

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BACKGROUND There are limited published data defining complete pleural fluid analysis, echocardiographic characteristics, or the presence or absence of ascites on sonographic or CT imaging in patients with hepatic hydrothorax. METHODS We reviewed pleural fluid analysis and radiographic, sonographic,

Study on correlation between liver ultrasonic appearance of patients with chronic hepatitis B and cirrhosis and hydrothorax.

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The aim of this study is to analyze the correlation between liver ultrasonic quantitative detection indexes of patients with chronic hepatitis B and cirrhosis and hepatic hydrothorax. We selected 56 cases of patients with chronic hepatitis B and cirrhosis combined with hepatic hydrothorax in our

Effects of beta-adrenergic blockade or stimulation on net rate of hydrothorax absorption.

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We determined in anesthetised rabbits the net rate of liquid absorption (NRLA) from Ringer or 1% albumin-Ringer hydrothoraces with the beta-blocker propranolol (or nadolol) or the beta-agonist terbutaline. The beta-blocker reduced NRLA by 38% in 2 ml Ringer hydrothoraces, and did not change it in 2

A unilateral hydrothorax as the only manifestation of ovarian hyperstimulation syndrome: a case report.

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OBJECTIVE To describe a rare case of unilateral hydrothorax occurrence after ovarian stimulation for IVF. METHODS Case report. METHODS A university hospital. METHODS A 39-year-old female suffering from primary infertility due to a severe male factor. METHODS Thoracocentesis with IV albumin

Uterine leiomyoma associated with ascites and hydrothorax.

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Uterine leiomyoma associated with ascites and hydrothorax was apparently cured by total hysterectomy. This is believed to be the 15th recorded case of this type of Pseudo-Meigs' syndrome and the first in a patient with severe curvature of the spine and dorsolumbar scoliosis. The association with

Radioisotope scintigraphy in the diagnosis of hepatic hydrothorax.

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BACKGROUND Pleural effusion in cirrhotic patients (hepatic hydrothorax) may result from migration of ascitic fluid across defects in the diaphragm. Biochemical analysis of ascitic and pleural fluid provides only indirect information about the nature and origin of the effusion. The present study was

A case of hepatic hydrothorax.

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Hepatic hydrothorax occurred in a 38-year-old woman of severe hepatic failure complicated with pulmonary tuberculosis. The transdiaphragmatic passage of ascitic fluid to the pleural cavity was visually shown with a scintigram using technetium-99m macroaggregated albumin and also shown with a tracer

The use of the diagnostic radionuclide ascites scan to facilitate treatment decisions for hepatic hydrothorax.

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A 44-year-old man had an intractable right-sided pleural effusion due to cirrhosis, despite the absence of abdominal ascites. Instillation of Tc-99m macroaggregated serum albumin under CT guidance into the peritoneal space demonstrated transdiaphragmatic communication. This finding indicated the

Scintigraphic demonstration of a diaphragmatic defect as the cause of massive hydrothorax in cirrhosis.

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A 64-year-old man presented with a massive right-sided hydrothorax associated with cirrhosis of the liver. There was no clinical evidence of ascites nor other underlying disease. The usual complementary tests failed to demonstrate any causative abnormality. Chest and abdominal scintigraphy after

Indwelling Pleural Catheters in Hepatic Hydrothorax: A Single-Center Series of Outcomes and Complications.

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BACKGROUND Treatment of hepatic hydrothorax (HH) generally involves sodium restriction, diuretics, and serial thoracentesis. In more advanced cases, transjugular intrahepatic portosystemic shunt and liver transplantation may be required. Previously, indwelling tube drainage has been avoided due to

Management of massive and persistent ascites and/or hydrothorax after liver transplantation.

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OBJECTIVE To describe the results of the treatment of eight liver transplantation (LT) patients subsequently developing large volumes of long-lasting ascites. METHODS Between August 1996 and February 2003, 405 LTs were performed in 375 patients, eight (1.97%) of whom (six men and two women of mean

Refractory hepatic hydrothorax treated with transjugular intrahepatic portosystemic shunt.

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A 66-year-old cirrhotic woman was referred to our hospital for evaluation of refractory pleural effusion and dyspnea. Massive right sided-pleural effusion but no ascites was detected. She had been treated with diuretics and albumin, repeated thoracenteses, and pleural drainage with an intercostal

[Hemilateral hydrothorax and atelectasis during laparoscopic Nissen fundoplication].

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A 57-year-old woman, weighing 75 kg, with gastroesophageal sliding hernia, received laparoscopic Nissen fundoplication under general anesthesia. Although artificial ventilation was carried out uneventfully when the surgery started, sudden decreases in SpO2 and thoraco-pulmonary compliance were noted

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