hypervitaminosis a/potassium

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Changes in sodium and potassium excretion and urinary volume in rats submitted to hypervitaminosis A.

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Hypervitaminosis A induces the following changes in rat kidney: decrease in sodium and potassium excretion and an increase of urinary volume. In order to determine the possible reversibility of these alterations, the authors allowed the hypervitaminotic animal to recover for 30 and 60 days. After a

Hypervitaminosis A causing hypercalcemia in cystic fibrosis. Case report and focused review.

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Hypercalcemia is a rare complication of hypervitaminosis A. We report a pediatric patient with cystic fibrosis (CF) and pancreatic insufficiency who was found to have hypervitaminosis A causing hypercalcemia, complicated by nephrocalcinosis and renal impairment. The patient is a 4-year-old girl with

[Effect of acute hypervitaminosis A on serum concentrations of Na, K, Mg, Fe, Zn and Cu in rats].

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The effect of hypervitaminosis A on the content of sodium, potassium, magnesium, iron, zinc and copper in rat serum was studied. Results were compared to findings in non-treated animals. The serum content of potassium, magnesium and copper increased significantly, while the content of sodium, zinc

Ro 15-1570, a new sulfur-containing retinoid devoid of bone toxicity in rats.

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Repeated ingestion of high doses of retinoids cause the so-called hypervitaminosis A syndrome. In rats the main symptoms are weight loss, alopecia, erythema, desquamation of the skin, and alterations of the skeletal system, including bone fractures. In the present study, three retinoids (Ro 15-1570,

Effects of dietary counseling on vitamin A, B-1, B-2 and zinc status of chronic hemodialysis patients.

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Twenty-one chronic hemodialysis patients underwent nutritional evaluation and regular dietary counseling to improve the protein and energy intake for 8 weeks. As a result, the mean serum potassium and phosphorus concentrations were increased but were still within the normal ranges. After counseling,

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