hypokalemia/seizures

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[Encephalopathy, convulsions and hypopotassemia in theophylline poisoning: a case analysis].

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A 73-year-old man was admitted to our hospital after convulsive seizures preceded by encephalopathy lasting one week. He had a history of long-standing chronic obstructive pulmonary disease and analysis showed hypokalemia, respiratory alkalosis and toxic levels of theophylline. We discuss the

A 3-year old girl with seizures, hypokalemia and metabolic alkalosis.

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A 3-year-old girl presented to the emergency department with seizures, low-grade fever and vomiting. She had tachycardia and a slow capillary refill. Blood pressure could not be measured. Because of suspected sepsis and/or meningo-encephalitis, broad spectrum antibiotics and antiviral medication

[A young woman with anorexia, hypokalemia and convulsion].

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A woman in her twenties, who had vomited daily for a year, developed serious anorexia (BMI 14) and hypokalemia. She was admitted to a local hospital because of listlessness and palpitations. Blood tests showed pH 7.62 (7.35-7.45), pCO2: 5.51 kPa (4.70-6.00), and potassium 2.3 mmol/l (3.5-5.0), later

Recurrent aborted sudden cardiac death with seizures and rhabdomyolysis due to bulimia-induced hypokalemia: report of one case.

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Recurrent vomiting due to bulimia associated with abuse of furosemide and laxatives causing severe hypokalemia may result in recurrent aborted sudden cardiac death (SCD) and seizures. We report a 25-year-old female with a history of bulimia associated with abuse of furosemide and laxatives since the

AMSA-induced seizures in patients with hypokalemia.

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Levetiracetam-associated Hypokalemia and Hypomagnesaemia among Two Patients Treated for Seizures.

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Recurrent seizure-triggered Takotsubo associated with hypokalemia and hypomagnesemia.

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[Elderly patient on peritoneal dialysis with convulsions, dysautonomy, diarrhea and hypokalemia].

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Seizures following burns of the skin. III. Electroencephalographic recordings.

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1. EEGs were recorded on 40 burned (acute) patients during their hospitalization. Abnormal records were seen in 88% of the 49 records, all showing slow waves and nearly 10% with epileptiform activity. EEG abnormality seems related to the combination of total body surface burned and time after burn

[Electrolyte disorders, EEG changes and epileptic seizures in alcohol withdrawal delirium].

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For 180 patients suffering alcohol-withdrawal induced delirium, electrolytic concentration in the serum of Na, K, Ca, and Mg was determined in the early withdrawal phase, and the electroencephalograms of 95 delirium patients evaluated in respect of local and diffuse changes and epileptic activity,

Wilson's disease presenting with hypokalemia, hypoparathyroidism and renal failure.

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Wilson's disease (WD) is not as rare as once believed, and has a wide range of presentations with equally wide range of age of onset. Sometimes the primary presentation might be unusual and may require a thorough investigation to avoid a misdiagnosis. Our case presented with uncontrolled seizures,

[A case of pseudohypoparathyroidism (PHP) type II associated with Bartter's syndrome--restoration of phosphaturic response to parathyroid hormone (PTH) by treatment for hypopotassemia].

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We report a case of PHP Type II whose phosphaturic response to PTH was restored by treatment for complicated Bartter's syndrome. A 34-year-old woman was admitted to our hospital in July 1990 because of tetanic convulsion. The physical examination showed normal blood pressure (118/62mmHg), round face

Therapeutic hypothermia for refractory status epilepticus in a child with malignant migrating partial seizures of infancy and SCN1A mutation: a case report.

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Status epilepticus (SE) is a common indication for neurocritical care and can be refractory to standard measures. Refractory SE (RSE) is associated with high morbidity and mortality. Unconventional therapies may be utilized in certain cases, including therapeutic hypothermia (TH), bumetanide, and

Hypokalemia-induced abnormal movements: case report.

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BACKGROUND A sudden loss of consciousness followed by abnormal movements can be ictal or syncopal in origin. Transient response by the brain to sudden decrease of blood flow may cause sudden loss of consciousness followed by abnormal movements that mimic seizure. Dysrhythmia is one of the important

Gitelman Syndrome in a School Boy Who Presented with Generalized Convulsion and Had a R642H/R642W Mutation in the SLC12A3 Gene.

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An 8-year-old Japanese boy presented with a generalized convulsion. He had hypokalemia (serum K 2.4 mEq/L), hypomagnesemia, and metabolic alkalosis (BE 5.7 mmol/L). In addition, his plasma renin activity was elevated. He was tentatively diagnosed with epilepsy on the basis of the

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