hypokinesia/infarction

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Extension of hypokinesia into angiographically perfused myocardium in patients with acute infarction.

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OBJECTIVE This study was performed to determine whether left ventricular hypokinesia due to acute myocardial infarction lies between the site of coronary artery occlusion and the end of the infarct-related artery in patients. BACKGROUND Normalizing for the size of the risk region reduces variability

Acute myocardial infarction with normal and near normal coronary arteries. Documentation with coronary arteriography within 12 1/2 hours of the onset of symptoms in two cases (three episodes).

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Three instances (in two patients) of acute myocardial infarction associated with arteriographically normal or near normal coronary arteries are reported. One patient with a lateral infarction had a normal coronary arteriogram and hypokinesia of the lateral wall. Another patient had two infarctions:

Acute Epstein-Barr virus myocarditis simulating myocardial infarction with cardiogenic shock.

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We have reported the case of a 38-year-old white woman with substernal chest pain, hypotension, and ECG changes suggesting acute anterior myocardial infarction. Cardiac catheterization revealed no coronary artery pathology, but severe global hypokinesia was noted on left ventriculogram and

[32-year-old patient with acute myocardial infarction possibly induced by the appetite suppressant sibutramine].

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METHODS A 32-year-old, very slightly overweight woman (body mass index of 25) without any cardiovascular risk factors presented with acute chest pain. She reported taking the appetite suppressant sibutramine daily for three months. She was not pregnant and did not remember episodes of serious mental

[Detection of disease of the anterior interventricular artery by 2-dimensional echocardiography in acute inferior infarction. Comparison with the electrocardiographic data].

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The authors report their experience of 2D echocardiography in the acute stage of myocardial infarction. One hundred patients, 60 men and 40 women, aged 60 +/- 4.5 years (range 32 to 69 years) were admitted to hospital with an uncomplicated inferior myocardial infarction and underwent 2D

Role of myocardial perfusion scintigraphy post invasive coronary angiography in patients with Myocardial Infarction.

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BACKGROUND The presence of severe hypokinesia or akinesia and near complete stenotic lesions on coronary angiography, in a patient with acute myocardial infarction raises a question of viability in the involved territory and its response to revascularization. The decision of revascularization can be

Pump dysfunction after myocardial infarction: importance of location, extent and pattern of abnormal left ventricular segmental contraction.

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To delineate the relative effects on left ventricular function of the site, extent and nature of the abnormal left ventricular segmental contraction (dyssynergy) and thereby determine the mechanism by which anterior myocardial infarction results in greater depression of left ventricular performance

[Acute myocardial infarction triggered by salbutamol in asthma bronchiale].

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The authors report the case of an 77-year-old woman with no evidence of previous cardiac disease who developed a non-Q-wave myocardial infarction after beta2-mimetic treatment for the exacerbation of her chronic bronchial asthma. After the five dose of salbutamol spray she developed chest pain. The

Acute myocardial infarction associated with albuterol.

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OBJECTIVE To report a case of acute myocardial infarction (AMI) following the use of albuterol (salbutamol) in a patient without preexisting coronary artery disease and to review the related literature. METHODS An 84-year-old white woman with no history of cardiac disease was treated for an

[Non-transmural anterior wall infarct: changes in myocardial energy metabolism in remaining vital myocardium].

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To characterize energy metabolism in vivo after nontransmural anterior myocardial infarction, patients (group A, n = 19) with a stenosis of left anterior descending artery (LAD) and anterior wall hypokinesia by levocardiography were examined by phosphorus magnetic resonance spectroscopy (MRS).

[Hemiparkinsonism due to a solitary infarction of the right external segment of the globus pallidus: a case report].

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Three months prior to presentation, a 76-year-old woman suffered from insomnia and was prescribed some antidepressants and hypnotics. At that time, brain MRI showed no cerebral infarcts. Having developed an action tremor of the left hand, bradykinesia, and unstable gait, she visited our hospital.

Acute Myocardial Infarction during Induction Chemotherapy for Acute MLL t(4;11) Leukemia with Lineage Switch and Extreme Leukocytosis.

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BACKGROUND In patients with acute leukemias hemorrhage is the most frequent problem. Vein thrombotic events may appear rarely but arterial thromboses are exceptionally rare. We present a patient with acute leukemia and bilateral deep leg vein thrombosis who developed an acute myocardial infarction

Acute myocardial infarction and renal infarction in a bodybuilder using anabolic steroids.

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A 41-year-old male bodybuilder was admitted with acute inferior myocardial infarction. The patient had been using oxymetholone and methenolone to increase his performance for 15 years and quitted smoking three years before. He underwent successful primary percutaneous coronary intervention (PCI) and

[Heparin induced thrombocytopenia, left ventricular thrombus and cerebral embolism during an acute myocardial infarction].

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METHODS A 58-year-old male patient was admitted to the emergency room after reanimation in hypotensive shock, under sedation and respiration. METHODS The cardiac laboratory markers were within normal limits. The ECG demonstrated a sinustachycardia with typical infarction signs. The echocardiogram

The role of early measurement of nitrogen-13 ammonia uptake for predicting contractile recovery after acute myocardial infarction.

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Previous studies have shown that the maintenance of cell membrane integrity and metabolism requires the persistence of residual myocardial blood flow. The purpose of this study was to assess the role of N-13 ammonia positron emission tomographic (PET) imaging performed early after an acute

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