leukocytosis/protease

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Absence of plasma protease-antiprotease imbalance in the formation of saccular cerebral aneurysms.

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OBJECTIVE We examined the hypothesis that a plasma protease-antiprotease imbalance contributes to the formation of saccular cerebral aneurysms and the suggestion that the assay of these enzymes might be a screening tool for people at higher risk for aneurysm formation. METHODS From June 1997 through

New insights in the regulation of leukocytosis and the role played by leukocytes in septic shock.

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Leukocytosis is the result of white blood cell production in the bone marrow, margination onto the blood and lymph vessels and elimination of leukocytes in the peripheral tissues and the spleen. The regulation of the exit of leukocytes from the circulation at peripheral inflammatory sites is well

The Role of Neutrophil Proteases in LPS-Induced Production of Regulatory Rheumatoid Factor that Suppresses Autoimmunity.

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BACKGROUND We recently identified a regulatory rheumatoid factor (regRF), the production of which is associated with autoimmune disease resistance and remission. In studies of regRF in the blood of healthy rats, spontaneous increases in the regRF level were noted. We suggest that in the normal

Clinical evidence for the role of Trichomonas vaginalis in regulation of secretory leukocyte protease inhibitor in the female genital tract.

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BACKGROUND Secretory leukocyte protease inhibitor (SLPI) is responsible for regulating inflammatory damage to and innate and adaptive immune responses in the vaginal mucosa. Depressed cervicovaginal SLPI levels have been correlated with both Trichomonas vaginalis infection and poor reproductive

Cerebrospinal fluid viral escape in aviremic HIV-infected patients receiving antiretroviral therapy: prevalence, risk factors and neurocognitive effects.

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During antiretroviral therapy, HIV RNA can be detected in cerebrospinal fluid (CSF) when it is undetectable in plasma, a condition termed 'CSF viral escape'. The aim of the current study was to determine the prevalence and risk factors for CSF viral escape in two large cohorts in the

Multivariate analysis of risk factors for thrombus formation in University of Tokyo ventricular assist device.

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Of 77 University of Tokyo ventricular assist devices used in a total of 70 patients at 21 institutions, 13 pumps were found to have macroscopic thrombus formations. Because 19 devices that were used for less than 24 hours showed no thrombus deposition, they were considered not to have been

Abdominal aortic aneurysm, Leriche's syndrome, inguinal herniation, and smoking.

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We previously found an increase in serum proteolytic activity in smokers with direct inguinal herniation and a similar imbalance in smokers with abdominal aortic aneurysm (AAA), but not in smokers with Leriche's syndrome (LS). If the protease imbalance in the blood of smokers with AAA or herniation

Systemic inflammatory response syndrome is reduced by preoperative plasma-thrombo-leukocyte aphaeresis in a pig model of cardiopulmonary bypass.

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OBJECTIVE The systemic inflammatory response syndrome (SIRS) after cardiac surgery with cardiopulmonary bypass (CPB) exacerbates organ dysfunction and increases postoperative mortality. The aim of this study was to reduce SIRS after CPB in a pig model by profoundly decreasing all blood defence

[Retrospective and prospective studies of patients with Cogan I syndrome].

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Since August 1987 we documented the course of three patients with Cogan-I-Syndrome (CS) (interstitial keratitis and audio vestibular disorder). The patients (2 female, 1 male) received oral protease therapy in addition to the corticosteroids. Under oral treatment with proteases the corticosteroid

A rare and severe cutaneous adverse effect of telaprevir: drug rash with eosinophilia and systemic symptoms.

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Telaprevir is a specific inhibitor of the hepatitis C (HCV) serine protease 3. Cutaneous side effects have been reported with telaprevir. Drug reaction with eosinophilia and systemic symptoms (DRESS) is a rare yet severe adverse drug-induced reaction characterized by exfoliative dermatitis and

Neutrophil metabolic activity but not neutrophil sequestration reflects the development of pancreatitis-associated lung injury.

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OBJECTIVE Activated neutrophils are presumed to injure pulmonary endothelium by releasing oxygen radicals, proteases, and proinflammatory cytokines. Standard counting methods do not distinguish between leukocytosis, neutrophil sequestration, and activation. We used leukocyte uptake of the glucose

Viral escape in the CNS with multidrug-resistant HIV-1.

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BACKGROUND HIV-1 viral escape in the cerebrospinal fluid (CSF) despite viral suppression in plasma is rare [1,2]. We describe the case of a 50-year-old HIV-1 infected patient who was diagnosed with HIV-1 in 1995. Antiretroviral therapy (ART) was started in 1998 with a CD4 T cell count of 71 cells/ìL

Limited proteolysis of a chemically modified third component of human complement, C3, by cathepsin G of human leukocytes.

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We have investigated the limited proteolysis of the third component of complement, C3, by a human leukocyte protease, cathepsin G, by using a chemically modified C3, which was prepared by treatment of C3 with methylamine and a fluorescent thiol reagent, N-(dimethylamino-4-methylcoumarinyl)-maleimide

Symptomatic HIV CNS viral escape among patients on effective cART.

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The clinical syndrome in symptomatic HIV associated CNS viral escape is poorly defined. We attempted to describe the clinical syndrome, laboratory profile, radiological features and outcomes of HIV infected patients with symptomatic central nervous system (CNS) viral escape in our

Symptomatic CSF HIV Escape in Patients on Anti Retroviral Therapy - A Case Series from India

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The central nervous system is believed to be a safe sanctuary site which is established very early in the course of HIV infection where the virus can escape antiretroviral therapy. HIV associated neurocognitive disorder (HAND) is seen to occur even in patients on successful systemic ART. HIV RNA can

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