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lignin/hemorrhage

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Organ distribution and toxicity of lignin.

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It is suggested that 125I-labeled natural and synthetic lignins administered orally to mice are absorbed through the digestive tract and excreted via urine, in addition to feces. Intravenous administration of lignins produced preferential accumulation of radioactivity in the liver and also in the

[The prevention of the translocation of intestinal microflora after the rescue of an organism from a terminal state].

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In experiments on guinea pigs, the animals were resuscitated from clinical death caused by the acute loss of blood and subsequently treated intragastrically with enterosorbents: activated carbon fibrous material, alone and in combination with polymyxin B, polyphepan (a lignin derivative),

Conservative treatment of haemorrhoids.

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1. INTRODUCTION Hemorrhoids are vascular cushions underlying the distal rectal mucosa and contributing for approximately 15-20% of the resting anal pressure with a complete closure of the anal canal. They can become pathological (hemorrhoidal disease, HD) being the most common cause of painless

Allosterism-based simultaneous, dual anticoagulant and antiplatelet action: allosteric inhibitor targeting the glycoprotein Ibα-binding and heparin-binding site of thrombin.

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BACKGROUND Allosteric inhibition is a promising approach for developing a new group of anticoagulants with potentially reduced bleeding consequences. Recently, we designed sulfated β-O4 lignin (SbO4L) as an allosteric inhibitor that targets exosite 2 of thrombin to reduce fibrinogen cleavage through

Pseudomonas syringae pv. actinidiae: a re-emerging, multi-faceted, pandemic pathogen.

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Pseudomonas syringae pv. actinidiae is the causal agent of bacterial canker of green-fleshed kiwifruit (Actinidia deliciosa) and yellow-fleshed kiwifruit (A. chinensis). A recent, sudden, re-emerging wave of this disease has occurred, almost contemporaneously, in all of the main areas of kiwifruit
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