lyme disease/proline

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CD14 signaling reciprocally controls collagen deposition and turnover to regulate the development of lyme arthritis.

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CD14 is a glycosylphosphatidylinositol-anchored protein expressed primarily on myeloid cells (eg, neutrophils, macrophages, and dendritic cells). CD14(-/-) mice infected with Borrelia burgdorferi, the causative agent of Lyme disease, produce more proinflammatory cytokines and present with greater

Identification of the determinant conferring permissive substrate usage in the telomere resolvase, ResT.

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Linear genome stability requires specialized telomere replication and protection mechanisms. A common solution to this problem in non-eukaryotes is the formation of hairpin telomeres by telomere resolvases (also known as protelomerases). These enzymes perform a two-step transesterification on

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