methyl palmitate/obesity

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The effects of methyl palmitate, a putative regulator from perivascular fat, on the contractility of pregnant human myometrium.

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OBJECTIVE Methyl palmitate is thought to cause relaxation in vascular smooth muscle by opening voltage-activated potassium channels. We have tested the hypothesis that methyl palmitate, a putative regulator from perivascular fat, is an inhibitor of the contractility of human pregnant myometrium and

The metabolome profiling and pathway analysis in metabolic healthy and abnormal obesity.

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OBJECTIVE Mechanisms of the development of abnormal metabolic phenotypes among obese population are not yet clear. In this study, we aimed to screen metabolomes of both healthy and subjects with abnormal obesity to identify potential metabolic pathways that may regulate the different metabolic

The role of perivascular adipose tissue in obesity-induced vascular dysfunction.

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Under physiological conditions, perivascular adipose tissue (PVAT) attenuates agonist-induced vasoconstriction by releasing vasoactive molecules including hydrogen peroxide, angiotensin 1-7, adiponectin, methyl palmitate, hydrogen sulfide, NO and leptin. This anticontractile effect of PVAT is lost

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