panic disorder/cannabis

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Marijuana precipitation of panic disorder with agoraphobia.

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We report the case of a 16-year-old adolescent with the onset of a panic disorder with agoraphobia after a first panic attack during marijuana intoxication. There was a good response to standard cognitive behavioural therapy for panic disorder.

Comorbid cannabis use and panic disorder: short term and long term follow-up study.

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OBJECTIVE The aim of the study was to compare the treatment of panic disorder in patients with or without cannabis use according to response, relapse and side effects. METHODS 66 panic disorder (PD) patients were included in our study. All the subjects met the DSM-IV diagnosis of panic disorder

Association of cannabinoid receptor genes (CNR1 and CNR2) polymorphisms and panic disorder.

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Background and objectives: Panic disorder (PD) is an anxiety disorder characterized by recurrent and unexpected panic attacks along with sudden onset of apprehension, fear or terror. The endocannabinoid system (ECS) has a role in stress recovery, regulating anxiety. The aim of this study was

Comparison of Cortisol Stress Response in Patients with Panic Disorder, Cannabis-Induced Panic Disorder, and Healthy Controls.

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Little research effort has so far been dedicated to the analysis of the hypothalamic-pituitary-adrenal axis of aetiologically differing subgroups of patients with panic disorder (PD). The current study aimed at a deeper understanding of the cortisol stress response in cannabis-induced

Marijuana and panic disorder.

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Marijuana (cannabis) use is anecdotally said to precipitate anxiety symptoms in patients with panic disorder. Is there any research evidence to support this? Also, can marijuana use precipitate or expose paranoia in patients with an underlying bipolar disorder?

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[Sociodemographic profiles, addictive and mental comorbidity in cannabis users in an outpatient specific setting].

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BACKGROUND In the 1990s, cannabis consumption in France increased considerably. So, in 10 years, the number of adolescents reporting regular cannabis use (10 or more times during the last 12 months) tripled. In 2004, an official program to address problems related to cannabis addiction was

Cannabinoid modulation of predator fear: involvement of the dorsolateral periaqueductal gray.

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The present study investigated the effects of systemic or intra-dorsolateral periaqueductal gray (dlPAG) administration of CB1 agonists on behavioural changes induced in rats by predator (a live cat) exposure, a model of panic responses. Since nitric oxide (NO) and cannabinoid neurotransmission are

Correlates and 6-month outcomes for co-occurring cannabis use in rural and urban at-risk drinkers.

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We know little about the functional correlates of recent cannabis use when such use is additional to an "alcohol disorder" in non-treatment populations. We report on data from a prospective study of a large probability community survey of 733 at-risk drinkers in six Southern U.S. states (Alabama,

Psychosis risk syndrome comorbid with panic attack disorder in a cannabis-abusing patient affected by Arnold-Chiari malformation type I.

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OBJECTIVE An 18-year-old man with Arnold-Chiari malformation (ACM) type I developed sudden panic attacks. He also manifested sleep disorder, cannabis abuse, and psychosis-risk syndrome (PRS). Although with average-superior intelligence, he had executive dysfunction. This prompted us to explore the

[Comorbidity in 207 cannabis users in a specific outpatient setting].

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BACKGROUND Health care seeking for a problematic use of cannabis is in progress in France. OBJECTIVE The aim is to assess the addictive and psychiatric comorbidity in cannabis users seen in the specific setting at the Lariboisière hospital. METHODS Two hundred and seven cannabis users were included

Cannabis expectancies in substance misusers: French validation of the Marijuana Effect Expectancy Questionnaire.

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The aim of this study was to evaluate the psychometric properties of the French version of the Marijuana Effect Expectancy Questionnaire (48 items) and study the cannabis expectancies according to the patterns of substance use and psychiatric disorders (DSM-IV). A sample of 263 subjects (average age

Marijuana use and panic psychopathology among a representative sample of adults.

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This study examined the relations between marijuana use and panic attacks and panic disorder using a large representative survey of adults (N = 5,672; 53% women; M(age) = 45.05 years, SD = 17.9) conducted in the United States (Kessler et al., 2004). After adjusting for sociodemographic variables

Effects of alprazolam and cannabinoid-related compounds in an animal model of panic attack.

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Selective stimulation of carotid chemoreceptors by intravenous infusion of low doses of potassium cyanide (KCN) produces short-lasting escape responses that have been proposed as a model of panic attack. In turn, preclinical studies suggest that facilitation of the endocannabinoid system attenuate

Cannabinoid CB1 receptors mediate the anxiolytic effects induced by systemic alprazolam and intra-periaqueductal gray 5-HT1A receptor activation.

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The endocannabinoid system has been implicated in the modulation of behaviors related to anxiety and panic disorders. Accordingly, facilitation of CB₁ receptor signaling reduces the consequences of aversive stimuli in animal models. However, the role of the CB₁ receptor in the

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