persistent hyperplastic primary vitreous/resin

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High frequency of persistent hyperplastic primary vitreous and cataracts in p53-deficient mice.

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In order to investigate whether the p53 gene product plays a role in normal eye development, age matched p53-deficient mice and wild-type controls were sacrificed from day 2 to day 21 after birth. Eyes were paraffin-embedded and sectioned. Serial sections were taken at the level of the tunica

Early Postnatal Hyperoxia in Mice Leads to Severe Persistent Vitreoretinopathy.

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To describe a mouse model of hyperoxia-induced vitreoretinopathy that replicated some of the clinical and pathologic features encountered in infants with severe retinopathy of prematurity and congenital ocular conditions such as persistent hyperplastic primary vitreous. Experimental mice (C57BL/6J)

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