porphyrias/carbohydrate

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[Carbohydrate metabolic disorders in porphyria cutanea tarda].

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The frequency of diabetes mellitus was studied in all 150 registered patients suffering from porphyria cutanea tarda in Bulgaria. To 63 of the patients an oral glucose tolerance test was performed according to the criteria of the Diabetic Committee of the WHO (1979). Diabetes mellitus was found in 9

Carbohydrate metabolism in porphyria cutanea tarda.

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A study on carbohydrate metabolism (glycaemia and insulinaemia curves during the oral and the intravenous glucose tolerance tests) in 20 porphyria cutanea tarda patients revealed diabetes mellitus in 2 and impaired glucose tolerance in 1 patient, but the carbohydrate tolerance did not differ greatly

Carbohydrate metabolism in porphyria cutanea tarda.

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Porphyria cutanea tarda (PCT) has a known increased incidence of diabetes mellitus and hepatic involvement. We investigated glucose tolerance and glucoregulatory hormone alterations in seven patients with PCT and correlated these results with hepatic histology by percutaneous liver biopsy. Abnormal

[Carbohydrate metabolism and plasma insulin levels in 2 patients with intermittent acute porphyria].

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[ON CARBOHYDRATE METABOLISM IN PORPHYRIA CUTANEA TARDA].

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[Carbohydrate turnover and capillary changes in porphyria cutanea tarda].

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Treatment options in acute porphyria, porphyria cutanea tarda, and erythropoietic protoporphyria.

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The porphyrias are a group of uncommon metabolic diseases caused by enzyme deficiencies within heme biosynthesis that lead to neurotoxic or phototoxic heme precursor accumulation. There are four acute porphyrias characterized by neuropsychiatric symptoms: acute intermittent porphyria, variegate

Seizure management in acute hepatic porphyria: risks of valproate and clonazepam.

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Seizures may occur in acute intermittent porphyria or other hepatic porphyrias. Management is difficult, because barbiturates and hydantoins exacerbate the porphyric state. We studied one patient with major motor seizures and acute intermittent porphyria. The seizure disorder was exacerbated by

Acute intermittent porphyria: clinical and selected research aspects.

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Acute intermittent porphyria is an inborn error of metabolism characterized by the excretion of excess porphyrin precursors (porphobilinogen and usually delta-aminolevulinic acid) in the urine, and by sporadic attacks of neurologic dysfunction. The disease is complex, involving variable patterns of

[Nephrologists and porphyrias].

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As usually occurs for rare diseases, the word "PORPHYRIA" often recalls a confused topic with shaded boundaries, presenting "bullous" skin lesions, rare opportunity of diagnosis in clinical practice, unknown pathogenesis, and almost absent therapeutic options. The goal of this review is to draw

Changes in some intestinal enzyme activities in experimental hexachlorobenzene-induced porphyria and modifying effects of diet.

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Some diets exert a considerable influence on porphyrin metabolism and induction of microsomal liver enzymes in experimental porphyria induced by hexachlorobenzene (HCB). As HCB and its metabolites come into direct contact with intestinal mucosa, this study investigated the changes in the activities

The porphyrias.

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The heterogeneous group of diseases called the porphyrias may all be characterised by derangement of specific stages in the haem biosynthetic pathway. In the acute porphyrias; acute intermittent porphyria, urophorphyrinogen 1 synthase, hereditary coproporphyria, coproporphyrinogen oxidase and

Acute intermittent porphyria: case report and review of the literature.

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Acute intermittent porphyria is an unusual pathology with potentially severe consequences when not early detected. Among the possible causes of porphyric crises decrease of caloric intake has been described. A case of acute intermittent porphyria in the late postoperative period of a bariatric

Neuropathic pain in a patient with porphyria. Case report.

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OBJECTIVE Porphyrias represent a group of inherited or acquired disorders that involve enzymes that participate in heme synthesis. Acute manifestations affect the nervous system resulting in abdominal pain, vomiting, acute neuropathy, seizures, and mental disorders. The physiopathogeny results from

Oxidative stress in acute intermittent porphyria and lead poisoning may be triggered by 5-aminolevulinic acid.

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Highly reactive oxyradicals can be generated in vitro by iron-catalyzed aerobic oxidation of synthetic and naturally occurring substances capable of enolization in aqueous medium. Of biological interest are alpha-hydroxy- and alpha-aminocarbonyls such as carbohydrates, 5-aminolevulinic acid, and

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