purpura fulminans/neoplasms

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The surgical strategy of Purpura fulminans triggered by pyothorax associated with lung cancer.

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Purpura fulminans is a rare disease that usually causes sepsis and is accompanied by disseminated intravascular coagulation and symmetric gangrene of distal extremities. We had to consider the most appropriate surgery approach. The most important point was attempting to rescue the patient's ability

[Fibrinolytic purpura in cancer of the stomach].

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[Purpura fulminans in the newborn. Report of two cases successfully treated with heparin and antithrombin III].

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Purpura fulminans is a rare form of disseminated intravascular coagulation characterized by rapidly progressive purpuric lesions, hypotension and, in some cases, fever. In neonates, purpura fulminans usually develops following deficiency of anticoagulant protein C or S, although acquired forms have

Fatal group A Streptococcus purpura fulminans in a child receiving TNF-α blocker.

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Inhibition of tumor necrosis factor alpha (TNF-α) is effective in the treatment of many pediatric autoimmune diseases and inflammatory conditions. Commonly available biologic agents blocking TNF-α are infliximab, etanercept, and adalimumab. These agents have changed the management of rheumatic

Nose and upper lip reconstruction for purpura fulminans.

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BACKGROUND Purpura fulminans (PF) is a rare syndrome of intravascular thrombosis and haemorrhagic infarction of the skin. The initial symptom of PF is peripheral purpura which progresses to necrosis very rapidly. The prognosis of PF is poor, and the mortality is reported to be around 40%. Even if

Acute Purpura Fulminans - a rare cause of skin necrosis: A single institution clinicopathological experience.

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Purpura fulminans, an uncommon syndrome of intravascular thrombosis with hemorrhagic infarction of the skin, is often accompanied by disseminated intravascular coagulation (DIC), multi-organ failure and may ultimately lead to death.Herein we document 13

Presentation and outcome of purpura fulminans associated with peripheral gangrene in 12 patients at Mayo Clinic.

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BACKGROUND Dermatologists may be called to assist in the diagnosis and management of purpura fulminans. METHODS This retrospective case series details the clinical presentation and outcomes of patients presenting with purpura fulminans associated with peripheral gangrene between 1989 and

Acute infectious purpura fulminans: pathogenesis and medical management.

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Purpura fulminans (PF) is a potentially disabling and life-threatening disorder characterized by acute onset of progressive cutaneous hemorrhage and necrosis, and disseminated intravascular necrosis. Acute infectious PF occurs most commonly in the setting of meningococcemia due to elaboration of

The color of skin: purple diseases of the skin, nails, and mucosa.

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The color purple can be seen in several types of eruptions including inflammatory dermatoses like lichen planus, infectious dermatoses like ecthyma gangrenosum, neoplasms like Kaposi sarcoma, and vasculitis and vasculopathy. The current review focuses on the clinical appearance, pathophysiology, and

Protein C anticoagulant pathway and its role in controlling microvascular thrombosis and inflammation.

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OBJECTIVE To review the physiologic and biochemical mechanisms that suggest that protein C and activated protein C (APC) have unique properties that make them good candidates for the treatment of microvascular thrombosis, disseminated intravascular coagulation, and sepsis. METHODS A summary of

Prothrombotic and Proinflammatory Activities of the β-Hemolytic Group B Streptococcal Pigment.

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A prominent feature of severe streptococcal infections is the profound inflammatory response that contributes to systemic toxicity. In sepsis the dysregulated host response involves both immunological and nonimmunological pathways. Here, we report a fatal case of an immunocompetent healthy female

Coexistence of Kasabach-Merritt Syndrome and placental chorioangioma in a premature infant.

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Kasabach-Merritt syndrome is a rare life-threatening clinical presentation in neonatal period. it is characterized by giant hemangioma and serious thrombocytopenia. The diagnostic criteria include: 1) hemangiomas on skin, 2) thrombocytopenia or coagulopathy, 3) hemangioma on internal organs

Superantigen natural affinity maturation revealed by the crystal structure of staphylococcal enterotoxin G and its binding to T-cell receptor Vbeta8.2.

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The illnesses associated with bacterial superantigens (SAgs) such as food poisoning and toxic shock syndrome, as well as the emerging threat of purpura fulminans and community-associated methicillin-resistant S. aureus producer of SAgs, emphasize the importance of a better characterization of SAg

Warfarin-induced necrosis of the skin.

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Warfarin (Coumadin)-induced necrosis of the skin is an unusual condition associated with the administration of warfarin. The necrotic lesions appear suddenly and are extremely painful. Single or multiple lesions develop in a distinct pattern. A hypercoagulable state created early in the course of

Diagnosis and management of disseminated intravascular coagulation: the role of heparin therapy.

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Disseminated intravascular coagulation (DIC) is caused by a variety of underlying disorders, and criteria for diagnosis are not well defined. However, the most helpful are a low platelet count, positive plasma protamine test, and fibrinogen and fibrin degradation product levels viewed in the context

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