purpura/atrophy

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Thrombotic thrombocytopenic purpura with optic disk neovascularization, vitreous hemorrhage, retinal detachment, and optic atrophy.

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A 33-year-old woman had thrombotic thrombocytopenic purpura. She developed optic atrophy and bilateral neovascularization of the disk with extensive vitreous hemorrhage that progressed to traction and rhegmatogenous retinal detachment.

Cerebellar degeneration and immune thrombocytopenic purpura associated with human immunodeficiency virus infection (HIV).

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Cerebellar disorders associated with HIV infection are usually caused by opportunistic infections, central nervous system lymphoma, and toxic effects of medicines, nutritional and metabolic disorders, and cerebrovascular disease. We present an unusual association of cerebellar degeneration and

[Hematological evaluation of megakaryocytic vacuolar degeneration in patients with idiopathic thrombocytopenic purpura].

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Variable degrees of vacuolar degeneration of the cytoplasm of megakaryocytes(MEG) have been recognized in patients with idiopathic thrombocytopenic purpura(ITP). It has been questioned whether this degeneration is specific to the disease, as it is not seen in all patients, and few reports have

Primary biliary cirrhosis presenting as idiopathic thrombocytopenic purpura with deterioration after splenectomy.

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A case of primary biliary cirrhosis (PBC) presenting as idiopathic thrombocytopenic purpura is described. The patient, a 59-year-old woman, had, at the time when thrombocytopenia was first observed, an asymptomatic PBC. She responded only temporarily to corticosteroids, and splenectomy had to be

Deterioration of gas exchange in patients with severe thrombotic thrombocytopenic purpura with respiratory failure during therapeutic plasma exchange.

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Therapeutic plasma exchange (TPE) is a procedure performed on patients suffering from various disorders, including thrombotic thrombocytopenic purpura (TTP). As we noted a frequent transient deterioration in respiratory function when the procedure was performed on intensive care unit (ICU) patients,

Palatal Petechiae in the Absence of Group A Streptococcus in Pediatric Patients with Acute-Onset Neuropsychiatric Deterioration: A Cohort Study.

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BACKGROUND Palatal petechiae are 95% specific for streptococcal pharyngitis. Despite this, and despite prior research demonstrating that Group A Streptococcus (GAS) is a common antecedent to pediatric acute-onset neuropsychiatric syndrome (PANS) episodes, we anecdotally observed a low rate of

Apoptotic myocardial degeneration in thrombotic thrombocytopenic purpura.

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The objective of this study was to determine whether the known myocardial degeneration in TTP is due to apoptosis. In TTP the heart is often involved, including the cardiac conduction system. Despite many platelet occlusions of small coronary arteries, there is little myocardial necrosis. Why the

Case of Fatal Purpura Associated with Waxy Degeneration of the Striated Muscles, and Also of the Vessels in the Affected Parts.

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Massive subretinal haemorrhage secondary to age-related macular degeneration in a patient with idiopathic thrombocytopenic purpura.

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[Thrombopenia and optic nerve atrophy in Schoenlein-Henoch's purpura. A case with a fatal outcome].

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Purpura and cutaneous atrophy as side effects of inhaled corticosteroids in asthma therapy: a review.

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[Atrophy of the cerebellar granular layer in acute thrombopenic purpura].

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[The triad: diffuse senile atrophy, Bateman's purpura, spontaneous stellar pseudo-cicatrices. Association frequently encountered in cutaneous senescnence].

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Glomerulocystic kidney disease (GCKD) associated with Henoch-Schoenlein purpura: a case report and a review of adult cases of GCKD.

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We report on a 71-year-old male with Henoch-Schoenlein purpura (HSP) who developed glomerulocystic kidney disease (GCKD) without congenital abnormality. He had mild renal dysfunction. Renal biopsy findings showed mild proliferation of mesangial cells and matrixes, and tubular atrophy, interstitial

Linear atrophy and vascular fragility following ultrasoundguided triamcinolone injection for DeQuervain tendonitis.

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A 64 year-old woman presented with a one-yearhistory of purpuric, atrophic, linear patches alongthe left lateral forearm. The patient had receivedtwo ultrasound-guided triamcinolone injectionsone year earlier into her left extensor pollicis brevisand abductor pollicis longus tendon sheathsfor

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