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pyelonephritis/mirabilis
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Influence of pili on the virulence of Proteus mirabilis in experimental hematogenous pyelonephritis.
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The ability of heavily and lightly piliated Proteus mirabilis to infect the renal parenchyma was compared in a model of hematogenous pyelonephritis. Cortical abscesses occurred in 13 of 24 rats injected with lightly piliated P. mirabilis but in none of 24 rats challenged with heavily piliated
Contribution of Proteus mirabilis urease to persistence, urolithiasis, and acute pyelonephritis in a mouse model of ascending urinary tract infection.
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Proteus mirabilis, a significant cause of bacteriuria and acute pyelonephritis in humans, produces urease. This high-molecular-weight, multimeric, cytoplasmic enzyme hydrolyzes urea to ammonia and carbon dioxide. To assess the role of urease in colonization, urolithiasis, and acute pyelonephritis in
Protection against experimental Proteus mirabilis pyelonephritis in rats and significance of immunity.
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Intraperitoneal immunization with formalin-killed bacteria as well as previous hematogenous infection with Proteus O3H1 showed a protective effect against hematogenous pyelonephritis in rats when the homologous strain was used. Transfer of hyperimmune antisera protected against hematogenously
Efficacy of a Proteus mirabilis outer membrane protein vaccine in preventing experimental Proteus pyelonephritis in a BALB/c mouse model.
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A BALB/c mouse model of nonobstructive, ascending Proteus mirabilis pyelonephritis was characterized bacteriologically, histologically, and serologically from 3 to 28 days. Intravesicular administration of 2 X 10(8) P. mirabilis K7 resulted in the septic death of 9 (16%) of 57 mice by day 15. Among
Renal lysozyme levels in animals developing Proteus mirabilis-induced pyelonephritis.
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In animals developing experimentally induced unilateral pyelonephritis, both the infected kidney (IK) and the contralateral noninfected kidney (NIK) showed an immediate increase in renal lysozyme activity of about 5 days' duration after the unilateral injection of viable Proteus mirabilis into the
Experimental pyelonephritis induced by L-forms of Proteus mirabilis in rats.
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Pyelonephritis was produced in 75 white rats by applying a 24-hour ligature on the ureter and by intravenous injection of a suspension of 5X10(9) cells of a stable Proteus mirabilis L-form. Pyelonephritis was proved microscopically in 44% of the animals. The revertants of the introduced L-form play
Isolation of a Capnophilic and Extended-Spectrum β-Lactamase-Producing Proteus mirabilis Strain from the Urine of an Octogenarian Male Patient with Acute Pyelonephritis.
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A capnophilic Gram-negative rod-shaped bacterium was recovered from the urine of an octogenarian male patient with acute pyelonephritis. The isolate was found to produce CTX-M-2-type extended-spectrum β-lactamase. Interestingly, the isolate failed to grow on modified Drigalski (BTB) and MacConkey
[PYELONEPHRITIS AND SEPTICEMIA DUE TO PROTEUS MIRABILIS. TERMINAL CARDIOVASCULAR COLLAPSE].
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[Cell-mediated immunity in pyelonephritis: Passive transfer of adoptive immunity to retrograde Proteus mirabilis pyelonephritis in the rat (author's transl)].
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[Electrolyte disorders in infants with Proteus mirabilis pyelonephritis].
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[Apropos of acute pyelonephritis caused by Proteus mirabilis].
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[Host defense mechanism in pyelonephritis--activation of peritoneal macrophage and its protective effect in the rat with retrograde Proteus mirabilis pyelonephritis].
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ANTIBIOTIC THERAPY IN ESTABLISHED CHRONIC EXPERIMENTAL PROTEUS MIRABILIS PYELONEPHRITIS.
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[Experimental pyelonephritis from Proteus mirabilis L forms in rats].
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Nutritional iron status and susceptibility to Proteus mirabilis pyelonephritis in the rat.
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