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Emodin inhibits TNF alpha-induced MMP-1 expression through suppression of activator protein-1 (AP-1).
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Matrix metalloproteinases (MMPs) are the proteases involved in the degradation of the extracellular matrix. MMP-1 is thought to be one of the key enzymes acting in fibrolysis, a process closely related to tissue remodeling. In this study, we found that emodin, an anthraquinone which has been
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