silibinin/diarrhea

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Mushroom poisoning--from diarrhea to liver transplantation.

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Mushroom poisoning from the genus Amanita is a medical emergency, with Amanita phalloides being the most common species. The typical symptoms of nausea, vomiting, abdominal pain, and diarrhea are nonspecific and can be mistaken for gastroenteritis. If not adequately treated, hepatic and renal

Protection by silibinin against Amanita phalloides intoxication in beagles.

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A single oral dose of the lyophilized deathcap fungus Amanita phalloides (85 mg/kg body wt) caused gastrointestinal signs of diarrhea, retching, and vomiting in beagles after a latent period of 16 hr. The pathologic lesions; the increases in serum transaminase (GOT, GPT), alkaline phosphatase, and

[Severe vomiting, diarrhea].

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Because of heavy vomiting and a watery diarrhoea after consumption of two Amanita phalloides mushroom taken in suicidal attempt this 31-year old female patient came to our emergency ward. The diagnosis being clear a therapy with stomach irrigation, substitution of fluid and electrolytes as well as

Challenges in the early diagnosis of patients with acute liver failure induced by amatoxin poisoning: Two case reports.

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BACKGROUND Acute liver failure (ALF) induced by amatoxin-containing mushrooms accounts for more than 90% of deaths in patients suffering from mushroom poisoning. However, due to the fact that most hospitals cannot identify the species of mushrooms involved, or detect amatoxins, the early diagnosis

Four patients with Amanita Phalloides poisoning.

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Mushroom poisoning by Amanita phalloides is a rare but potentially fatal disease. The initial symptoms of nausea, vomiting, abdominal pain and diarrhea, which are typical for the intoxication, can be interpreted as a common gastro-enteritis. The intoxication can progress to acute liver and renal

[The development of a toxic megacolon due to Amanita phalloides poisoning. A rare complication].

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METHODS A 71-year-old previously healthy man consumed a mushroom meal containing Amanita phalloides, which he had confused with Agaricus campestris. Approximately 8 hours later, typical gastrointestinal symptoms began with copious loss of fluids. Treatment with penicillin G was initiated followed by

Milk Thistle

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Milk thistle (Silybum marianum) contains silymarin which is a mixture of flavonolignans, mainly silibinin (also known as silybin), as well as silycristine, silydianin, quercetin and taxifolin.[1] Silymarin is a standardized preparation extracted from the fruits (seeds) of milk thistle. Milk thistle

Investigation and analysis of Galerina sulciceps poisoning in a canteen.

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BACKGROUND Guizhou Province in China has an abundant resource of wild mushrooms, including numerous poisonous species which contain various toxins. The mortality rate from wild mushroom poisoning has been high in this area in recent years. Galerina sulciceps is a dangerously toxic mushroom which can

[Mushroom poisoning with a long period of development].

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A group of 87 patients with the signs of poisoning with mushrooms with along period of incubation (t = 12.4 +/- 6.2 h) has been reported. Nausea, vomiting and diarrhea dominate in the clinical picture in the first phase and hepatic and/or renal insufficiency in the second phase. Forty-one patients

A study of high-dose oral silybin-phytosome followed by prostatectomy in patients with localized prostate cancer.

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BACKGROUND Silibinin is a polyphenolic flavonolignan derived from milk thistle (Silybum marianium) with anti-oxidant properties. The purpose of the current trial was to determine the tissue and blood effects of high-dose silybin-phytosome in prostate cancer patients. METHODS Subjects with localized

Legalon® SIL: the antidote of choice in patients with acute hepatotoxicity from amatoxin poisoning.

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More than 90% of all fatal mushroom poisonings worldwide are due to amatoxin containing species that grow abundantly in Europe, South Asia, and the Indian subcontinent. Many cases have also been reported in North America. Initial symptoms of abdominal cramps, vomiting, and a severe cholera-like

Hepatoprotective potential of chestnut bee pollen on carbon tetrachloride-induced hepatic damages in rats.

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Bee pollen has been used as an apitherapy agent for several centuries to treat burns, wounds, gastrointestinal disorders, and various other diseases. The aim of our study was to investigate the hepatoprotective effects of chestnut bee pollen against carbon tetrachloride (CCI4)-induced liver damage.

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