stomach ulcer/hypoxia

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Gastric ulcers induced by systemic hypoxia.

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OBJECTIVE to assess the effect of systemic hypoxia on gastric mucosa and the activation of stress-responsive transcription factors induced by hypoxia. METHODS in this experimental study, rats were allocated to control and experimental groups. The experimental group was divided into subgroups and

[The role of local factors regulating vascular blood flow in the development of hypoxia of the gastric mucosa in peptic ulcer].

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In 50 patients with gastric ulcer indices of oxygen tension (pO2) levels were investigated in correlation with the concentration of prostaglandins (E and F2 alpha), prostacyclin, thromboxane as well as with values of transmucous difference of potentials in different parts of the gastric mucosa, they

Relation of hypoxia-inducible factor-1alpha to vascular endothelial growth factor and vasoactive factors during healing of gastric ulcers.

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We studied the relation of hypoxia-inducible factor-1alpha (HIF-1alpha) to vascular endothelial growth factor and vasoactive factors during the healing of gastric ulcers. The gastric ulcers were divided into three stages (active stage, healing stage and scar stage). The expression of HIF-1alpha,

[Hypoxia as the common final cause of gastroduodenal ulcer].

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Hypoxia of abnormal physiologic origin as the final common pathway in gastroduodenal ulcer genesis.

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[Role of respiratory and hemic hypoxia in the pathogenesis of chronic gastritis and peptic ulcer in children].

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[Tolerance for moderate degrees of hypoxia of pilots suffering from peptic ulcer].

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[Morphological substrates in the perinatal anoxia syndrome. Intra-alveolar hemorrhage and hyaline membranes of the lung, multiple stomach ulcers, bilateral kidney cortex necroses and periventricular encephalomalacia in a newborn infant].

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[Differences in the behavior and resistance to stomach ulcer during stress in August and Wistar rats adapted and not adapted to hypoxia].

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[The influence of acute artificial hypoxia and of exertion on the acid secretion of the stomach in patients with gastroduodenal ulcer].

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Effect of hypoxia on the development of gastric ulcers in pylorus-ligated, reserpine-treated and immobilized rats.

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Sleep apnea and risk of peptic ulcer bleeding: a nationwide population-based study.

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OBJECTIVE Patients with sleep apnea sustain cessation of breath during sleep, leading to intermittent hypoxia, systemic inflammation, and sympathetic activation. These insults may contribute to initiation or progression of peptic ulcers. This retrospective matched-control cohort study explored the

Respiratory complications after upper abdominal surgery. The incidence of chest X-ray abnormalities, arterial hypoxemia and clinically recorded pulmonary complications.

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The incidence of postoperative chest X-ray abnormalities, arterial hypoxemia and clinically recorded pulmonary complications was studied in fifty-two 40-75-year-old men admitted for elective surgery for peptic ulcer or gallbladder disease. Twenty-eight (54%) patients had an abnormal chest X-ray and

Adaptation to hypoxia as a method of treatment and prevention of gastroduodenal mucosa lesions.

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Intermittent normobaric hypoxia (13-23 daily sessions 30-50 min each) alleviated the dyspeptic and pain syndromes, improved the objective characteristics of the gastrointestinal tract, and promoted healing of duodenal and peptic ulcers.

The clinical significance of postoperative chest X-ray abnormalities, arterial hypoxemia and clinically recorded respiratory complications.

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The clinical consequences of postoperative complications as defined by signs and symptoms, chest radiography and measurements of the arterial oxygen tension were studied in 53 men, aged 40-75, who were electively operated for peptic ulcer or gallbladder disease. Clinical signs of a respiratory

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