stomach ulcer/phosphatase

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[Changes in alkaline phosphatase activity and blood phosphorus levels in healthy persons and patients with peptic ulcer].

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Venous blood total phosphatase activity, its thermostable fraction, and inorganic phosphorus levels were measured thrice within 20-64 min in the course of registration of regular motor activity of the gastrointestinal tract in 38 men aged 18 to 57 (9 healthy and 29 with peptic ulcer). These

[Distribution and properties of the alkaline phosphatase in the human gastric mucosa in peptic ulcer].

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In duodenal ulcer alkaline phosphatase activity was the highest in mucosa of pyloric region of curvature ventriculi minor and it decreased distinctly in the tissues removed from pylorus. In cases of gastric ulcer, the enzymatic activity was high throughout the curvatura ventriculi minor reaching the

Mice deficient in protein tyrosine phosphatase receptor type Z are resistant to gastric ulcer induction by VacA of Helicobacter pylori.

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The vacuolating cytotoxin VacA produced by Helicobacter pylori causes massive cellular vacuolation in vitro and gastric tissue damage in vivo, leading to gastric ulcers, when administered intragastrically. Here we report that mice deficient in protein tyrosine phosphatase receptor type Z (Ptprz,

[Lipid peroxidation, the enzyme antioxidative system and acid phosphatase content of the gastric mucosa in stomach ulcer].

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Lipid peroxidation, a state of the antioxidative system and activity of acid phosphatase were studied in mucosal membrane of 53 patients with ulcerous disease of stomach. Increase of the acid phosphatase activity in cytoplasm, activation of lipid peroxidation and inhibition of the antioxidative

[Serum phosphatase activity in parodontosis in patients with duodenal peptic ulcer].

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[Change in the blood picture, serum protein makeup and leukocyte phosphatase activity in peptic ulcer].

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[The behavior of alkaline phosphatase and nucleic acids in normal gastric mucosa, in peptic ulcer and in epithelial gastric tumors with special reference to the alkaline phosphatase and thymonucleic acid relationship].

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Isoenzymes of alkaline phosphatase in patients operated upon for peptic ulcer.

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[Histochemical study of phosphatase activity in gastroduodenal ulcer].

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[Alkaline phosphatase isoenzymes in peptic ulcer].

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Pulicaria crispa mitigates gastric ulcer induced by ethanol in rats: role of treatment and auto healing.

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Context: Stomach ulcers are the common gastrointestinal disorders worldwide. Objective: This study aimed to investigate the therapeutic impact of Pulicaria crispa aerial parts ethanol extract against gastric ulcer in rats. Materials and methods: Ulcer was induced by one

Management of Citrus sinensis peels for protection and treatment against gastric ulcer induced by ethanol in rats.

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Introduction: Stomach ulcer is one of the most prevalent disorders worldwide. The study was aimed to isolate and characterize the major polymethoxylated flavonoids in Citrus sinensis peels petroleum ether extract and investigate its protective and curative effect on gastric ulcer.

Effect of Cardiospermum halicacabum on ethanol-induced gastric ulcers in rats.

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Ethanol extract of Cardiospermum halicacabum Linn. (Sapindaceae), in a concentration dependant manner (200-600mg/kg) inhibited gastric ulcers induced by oral administration of absolute ethanol. Further, the extract administration to rats resulted in an increase in levels of gastric glutathione and a

[Influence of cattle cord blood fraction below 5 kD on biochemical parameters of blood in experimental chronic stomach ulcer in rats].

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Influence of cattle cord blood fraction (below 5 kD) on lipid peroxidation product content and alkaline phosphatase activity-in peripheral blood was studied on the experimental subchronic stomach ulcer model in rats. It has been shown that the fraction administrations normalize thiobarbituric-active

Effect of the IGF-1/PTEN/Akt/FoxO signaling pathway on the development and healing of water immersion and restraint stress-induced gastric ulcers in rats.

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Insulin-like growth factor 1 (IGF-1) and Akt [also known as protein kinase B (PKB)] proteins have been reported to exhibit gastroprotective effects by reducing water immersion and restraint stress (WRS)-induced gastric mucosal cellular apoptosis. To confirm whether the IGF-1/PTEN/Akt/FoxO signaling

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