tetany/necrosis

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Parathyroid necrosis and hypocalcemic tetany induced in rabbits by L-asparaginase.

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Fifty percent of New Zealand white rabbits became profoundly weak, had generalized seizures and died between 22 and 47 hours after an intravenous injection of 1000 IU/kg of L-asparaginase. The biochemical correlate of this syndrome is severe hypocalcemia associated with marked, single cell,

[Spontaneous necrosis of parathyroid adenoma].

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The authors describe a female patient with large parathyroid gland adenoma presenting with clinical symptoms of primary hyperparathyroidism and severe hypercalcaemia. Before planned surgical treatment the patient spontaneously developed clinical symptoms of tetany. Hypocalcaemia was confirmed by

Clinical and clinicopathologic features of transport tetany of feedlot lambs.

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Clinical and clinicopathologic features of transport tetany in 9 feedlot lambs were investigated. The lambs became recumbent within 10 days after arrival in the feedlot. The mean serum calcium concentration, mean magnesium concentration, and calcium:phosphorus ratio were significantly (P less than

Acute electrical burns.

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Electric current can damage an individual by thermal heating of the tissues; by disregulating autonomously functioning organ systems, such as the circulatory and respiratory systems; or by once-only or continuing stimulation of the nerves and striated muscles. In the first case, the result is

Primary hypoparathyroidism in dogs: a retrospective study of 17 cases.

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OBJECTIVE To evaluate the clinico-pathological findings, response to treatment and prevalence of complications in dogs with primary hypoparathyroidism. METHODS Retrospective study of 17 dogs presenting to the University of Melbourne Veterinary Clinical Centre and Murdoch University Veterinary

[Clinical analysis of 6 cases of Bartter syndrome].

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OBJECTIVE To summarize the clinical characteristics of Bartter syndrome and investigate its pathogenesis. METHODS The clinical data of 6 cases of Bartter syndrome at our hospital from November 2006 to May 2010 were analyzed retrospectively. RESULTS The onset age of Bartter syndrome was 13-35 years

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