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tularemia/glutathione
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Biochemical responses and oxidative stress in Francisella tularensis infection: a European brown hare model.
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BACKGROUND
The aim of the present study was to investigate biochemical and oxidative stress responses to experimental F. tularensis infection in European brown hares, an important source of human tularemia infections.
METHODS
For these purposes we compared the development of an array of biochemical
Tularemia progression accompanied with oxidative stress and antioxidant alteration in spleen and liver of BALB/c mice.
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Francisella tularensis is the causative agent of tularemia. It is an intracellular pathogen with the ability to survive within phagosomes and induce pyroptotic cell death. In this study, we attempted to prove whether oxidative imbalance plays a significant role in tularemia pathogenesis. In our
Glutathione provides a source of cysteine essential for intracellular multiplication of Francisella tularensis.
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Francisella tularensis is a highly infectious bacterium causing the zoonotic disease tularemia. Its ability to multiply and survive in macrophages is critical for its virulence. By screening a bank of HimarFT transposon mutants of the F. tularensis live vaccine strain (LVS) to isolate intracellular
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