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atropine/inflamación
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Effects of atropine on anterior chamber depth and anterior chamber inflammation after primary trabeculectomy.
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OBJECTIVE
To investigate the effects of postoperative atropine on central and peripheral anterior chamber depth and anterior chamber inflammation in patients undergoing primary trabeculectomy.
METHODS
Two separate groups of patients who were phakic without previous intraocular surgery undergoing
Crystal structures of the complexes of a group IIA phospholipase A2 with two natural anti-inflammatory agents, anisic acid, and atropine reveal a similar mode of binding.
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Secretory low molecular weight phospholipase A(2)s (PLA(2)s) are believed to be involved in the release of arachidonic acid, a precursor for the biosynthesis of pro-inflammatory eicosanoids. Therefore, the specific inhibitors of these enzymes may act as potent anti-inflammatory agents. Similarly,
Atropine reverses the antinociception of nonsteroidal anti-inflammatory drugs in the tail-flick test of mice.
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The nonsteroidal anti-inflammatory drugs (NSAIDs) clonixin, diclofenac, piroxicam, ketoprofen, meloxicam, and paracetamol induced antinociception after intraperitoneal or intrathecal administration in mice submitted to an acute thermal algesiometric test without inflammation (tail-flick).
Effects of atropine, acetylsalicylic acid, FPL 55712, and phentolamine on increased histamine responsiveness of cat lung strips under conditions of airway inflammation.
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Our recent in vitro studies on airway smooth muscles of cats with turpentine oil inflammation showed an increase of isometric tension of the lung strips to histamine application. This communication describes the effect of atropine, acetylsalicylic acid, FPL 55712, and phentolamine on the histamine
Small-bowel obstruction and the effects of lidocaine, atropine and hexamethonium on inflammation and fluid losses.
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BACKGROUND
The profuse fluid losses and morbidity of patients suffering from obstructive ileus are closely related to inflammatory changes in the obstructed bowel wall. Previous experimental studies have shown that use of steroids and NSAIDs can reduce fluid losses in obstructive ileus. In the
Bilateral vagotomy or atropine pre-treatment reduces experimental diesel-soot induced lung inflammation.
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To investigate the role of the vagus nerve in acute inflammatory and cardiorespiratory responses to diesel particulate (DP) in the rat airway, we measured changes in respiration, blood pressure and neutrophils in lungs of urethane anesthetized Wistar rats 6-h post-instillation of DP (500 microg) and
Effects of atropine and propranolol on lung inflammation in experimental envenomation: comparison of two buthidae venoms.
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BACKGROUND
Previous works had shown that scorpion venom induced neurotransmitter elevation and an inflammatory response associated with various anatomo-pathological modifications. The most dangerous scorpions species in Algeria responsible for these effects are Androctonus australis hector (Aah) and
Non-neuronal, but atropine-sensitive ileal contractile responses to short-chain fatty acids: age-dependent desensitization and restoration under inflammatory conditions in mice.
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Intestinal epithelial cells sense short-chain fatty acids (SCFAs) to secrete non-neuronal acetylcholine (ACh). However, the roles of luminalSCFAs and epithelialACh under normal and pathological conditions remain unknown. We examined ileal contractile responses toSCFAs at different ages and their
Atropine treatment modifies LPS-induced inflammatory response and increases survival.
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OBJECTIVE
To explore the effect that Atropine, a competitive antagonist for the muscarinic acetylcholine receptor (mAChR), has on the response to LPS.
METHODS
Eight-week-old, male, B6 mice.
METHODS
Mice were treated with Atropine prior to, or after LPS challenge.
METHODS
Survival was monitored and
Effects of atropine upon the chemistry of inflammation.
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An Examination under Atropine of the Refractive State of Eyes with Normal Vision (20/20), and which have never been Affected with Asthenopia or Inflammation.
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Hydrogen sulfide causes vanilloid receptor 1-mediated neurogenic inflammation in the airways.
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Hydrogen sulfide (H(2)S) is described as a mediator of diverse biological effects, and is known to produce irritation and injury in the lung following inhalation. Recently, H(2)S has been found to cause contraction in the rat urinary bladder via a neurogenic mechanism. Here, we studied whether
Methanol extract of Cola nitida ameliorates inflammation and nociception in experimental animals.
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Methanol extract of Cola nitida (MECN) was evaluated for its anti-inflammatory and analgesic activities using rats and mice. Inflammatory activity of MECN was assessed by carrageenan-induced paw oedema while analgesic activity was evaluated by acetic acid -induced writhing and formalin paw
[Repetitive instillations of cyclosporin A eyedrop induce pupillary constriction and anterior chamber inflammation-like reactions in rabbits].
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We attempted to clarify the mechanism of atropine-resistant miosis caused by repetitive instillations of cyclosporin A (CyA) eye drop in rabbits by investigating the possibility that the miosis may be attributable to CyA-induced contraction of the iris sphincter muscle, and that it may be resulting
Sex modulated effects of sarin exposure in rats: Toxicity, hypothermia and inflammatory markers.
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This work focused on sex differences in rats exposed to sarin. Females were found to be more sensitive to sarin toxicity (LD50 67 μg/kg) than males (88 μg/kg), showed less acute hypothermic effects than males (at 120 min post sarin, 3.1 ± 1.1 and 4.5 ± 1 °C decrease, respectively), but with a
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