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Página 1 desde 104 resultados
Effects of atropine, glycopyrrolate, and cimetidine on gastric secretions in morbidly obese patients.
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Gastric volume and pH were studied immediately after induction of anesthesia and endotracheal intubation in 40 morbidly obese patients undergoing gastric bypass procedures. Of 14 patients receiving intramuscular atropine (0.6 mg) preoperatively, 67% had gastric pH values < 2.5; 33% of these patients
Physical training in hyperplastic obesity. V. Effects of atropine on plasma insulin.
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Ten hyperplastic obese women were trained physically on an ad-libitum diet preventing them from losing weight. Plasma-insulin concentrations decreased during oral glucose-tolerance test (OGTT), an effect which was more clearly seen after atropine administration. After intravenous-glucose
Atropine sulfate for treatment of bradycardia in a patient with morbid obesity: what may happen when you least expect it.
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A 74-year-old morbidly obese man was scheduled for surgical repair of an incisional ventral hernia. Anaesthesia was induced with propofol and fentanyl, and maintained with desflurane. A second dose of fentanyl 0.2 mg, given before starting surgery, resulted in sinus bradycardia and mild decrease of
Efficacy and safety of a fixed-dose combination of D-norpseudoephedrine, triiodothyronine, atropine, aloin, and diazepam in obese patients .
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OBJECTIVE
A fixed-dose combination (FDC) of D-norpseudoephedrine, tri-iodothyronine, atropine, aloin, and diazepam is used in Mexico for the short-term treatment of obesity; however, its efficacy and safety have been scarcely studied. The aim of this study was to analyze the efficacy and safety of
Atropine improves insulin sensitivity in both lean and abdominally obese subjects.
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BACKGROUND
Dysregulated autonomic nerve activity may contribute to the development of type 2 diabetes. The aim of this study was to assess the effects of an anticholinergic agent, atropine, and a cholinergic agent, physostigmine, on insulin sensitivity in lean and abdominally obese
The heart rate response to atropine in uremic patients, obese subjects before and during fasting, and patients with other chronic illnesses.
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[INFLUENCE OF ATROPINE ON THE BEHAVIOR OF THE R.Q. AFTER GLUCOSE LOADING IN OBESE HUMANS].
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Gastrin release in obese Zucker rats.
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In this study, gastrin release in the obese Zucker rat was investigated by in vivo and in vitro experiments. Obese rats exhibited normal plasma gastrin levels at 3 weeks (preobese), were moderately hypergastrinemic at 3 months and severely hypergastrinemic at 5 months, compared to lean littermates.
[Insufficient sugammadex effect in an obese pregnant woman undergoing cesarean section under general anesthesia].
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A 32-year-old pregnant woman (height 162 cm, weight 86 kg, age of fetus 25 weeks) without preoperative complications underwent an emergent cesarean section under general anesthesia. She was intubated with a 7.0-mm tracheal tube 40 s after receiving rocuronium 0.93 mg x kg(-1) and thiamilal 375 mg.
Vagal mediation of corticotropin-releasing-factor-induced increase in insulinemia in lean and genetically obese fa/fa rats.
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The effects of intracerebroventricular (i.c.v.) administration of corticotropin-releasing factor (CRF) on plasma glucose and insulin levels were investigated in lean Zucker (FA/FA) rats; i.c.v. CRF induced a rapid (within 1 min), marked, but transient increase in insulinemia that was not accompanied
Early phase insulin infusion and muscarinic blockade in obese and lean subjects.
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The effect of early phase insulin on postprandial levels of insulin, C-peptide, glucose, and glucagon was investigated in lean (n = 10) and obese (n = 12) subjects. Subjects underwent four conditions during ingestion of a meal (600 kcal): 1) saline infusion; 2) 10-min insulin infusion simultaneously
Central glucocorticoid regulation of parasympathetic drive to pancreatic B-cells in the obese fa/fa rat.
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The effects of glucocorticoids on the insulin secretory response to an intravenous glucose load have been studied in lean (Fa/?) and obese fa/fa Zucker rats. The role of parasympathetic drive to the pancreatic B-cells was assessed as that component of the insulin secretory response that was blocked
Sudden bradycardia and asystole in an obese patient after spinal anaesthesia: successful resuscitation with inadvertent "pacing thumps.".
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A 45-year old morbidly obese man with diabetes and severe chest problem had sudden bradycardia and sinus arrest in the Recovery Ward after an uneventful spinal anaesthesia. He responded promptly to "attempts" at external cardiac message with each thump producing a QTS complex until Atropine could be
Insulin resistance in two animal models of obesity: A comparison of HISS-dependent and HISS-independent insulin action in high-fat diet-fed and Zucker rats.
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Normal postprandial insulin sensitivity depends on the action of the hepatic insulin sensitizing substance (HISS), which requires hepatic parasympathetic nerve activation. Since HISS action is impaired in several pathological models, including the genetically-modified obese Zucker rat (OZR), we
Selective muscarinic sensitivity in perfused pancreata of obese Zucker rats.
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Insulin secretion was evaluated in response to the muscarinic agonist, bethanechol, and to the antagonist, atropine, in three-month-old female homozygous lean (Fa/Fa) and obese (fa/fa) Zucker rats, using an in vitro pancreas perfusion. Three doses of bethanechol were used (0.5, 5 or 50 microM).
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