bronchopulmonary sequestration/albúmina

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Radionuclide study in pulmonary sequestration.

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A radionuclide hemodynamic study was helpful in demonstrating blood supply from the aberrant artery in pulmonary sequestration. Two regions of interest were set up in corresponding lower lung fields of a patient suspected of sequestration in the left lower lobe. Following i.v. injection of Tc-99m

[Use of scintigraph in the diagnosis of pulmonary sequestration].

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To evaluate scintigraphic findings in cases of pulmonary sequestration, perfusion lung scintigrams were obtained with Tc-99m macroaggregated albumin, and radionuclide angiocardiograms were obtained with Tc-99m pertechnetate in 5 cases of pulmonary sequestration. Perfusion lung scintigrams showed

Evidence for neutrophil-related acute lung injury after intestinal ischemia-reperfusion.

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Intestinal ischemia-reperfusion injury is a common and important clinical event associated with the activation of an endogenous inflammatory response. Some of the mediators of this response may be involved in the pathogenesis of multiple organ system failure. The purpose of this study was to

Vascular injury in isolated sheep lungs. Role of ischemia, extracorporeal perfusion, and oxygen.

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Extracorporeal perfusion of isolated sheep lungs with blood after 30 min of ischemia caused injury manifested by polymorphonuclear (PMN) leukocyte sequestration, pulmonary hypertension, thromboxane release, and increased pulmonary vascular permeability. To determine the roles of ischemia,

Complement and neutrophil-mediated injury of perfused rat lungs.

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It has previously been shown that systemic complement activation in rats leads to acute lung injury. We have now employed an ex vivo model of perfused rat lung which allows a more detailed examination of the role of circulating blood elements and pulmonary vascular pressure changes in the acute lung

TNF-alpha release in endotoxemia contributes to neutrophil-dependent pulmonary edema.

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We examined whether the generation of tumor necrosis factor (TNF-alpha) after lipopolysaccharide (LPS) challenge contributes to increases in lung vascular permeability and water content. Guinea pig lungs perfused at constant flow with Ringer-albumin solution (0.5 g/100 ml) were challenged for 120

Pulmonary vascular injury induced by hemorrhagic shock is mediated by P-selectin in rats.

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To investigate whether the P-selectin-mediated leukocyte adhesion to the endothelial cells is involved in pulmonary vascular injury after hemorrhagic shock, we examined the effect of an anti-P-selectin monoclonal antibody (MAb PB1.3) on the pulmonary accumulation of leukocytes and the subsequent

Pulmonary damage by Vibrio vulnificus cytolysin.

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Vibrio vulnificus is an estuarine bacterium that causes septicemia and serious wound infection. Cytolysin produced by V. vulnificus has been incriminated as one of the important virulence determinants of bacterial infection. Cytolysin (8 hemolytic units) given intravenously to mice via their tail

A new elastase inhibitor from Streptococcus pneumoniae protects against acute lung injury induced by neutrophil granules.

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A neutrophil elastase-inhibitor isolated from lysed pneumococcal cells, as well as trypsin-digest peptides derived from this factor, were tested for their ability to suppress acute lung injury in mice treated with human neutrophil granule extracts. Injury was assessed by measuring pulmonary

Pulmonary platelet sequestration is increased following monocrotaline pyrrole treatment of rats.

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111In-labeled platelets were used to study the localization and survival of circulating platelets at various times after a single, intravenous administration of 3.5 mg/kg monocrotaline pyrrole (MCTP) to rats. Lung injury, assessed from elevated lung weight, lavage fluid total protein and albumin

Effects of hyperoxia on local and remote microcirculatory inflammatory response after splanchnic ischemia and reperfusion.

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Splanchnic ischemia-reperfusion (I/R) causes tissue hypoxia that triggers local and systemic microcirculatory inflammatory responses. We evaluated the effects of hyperoxia in I/R induced by 40-min superior mesenteric artery (SMA) occlusion and 120-min reperfusion in four groups of rats: 1) control

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