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[Neonatal diarrhea due to congenital glucose-galactose malabsorption: report of seven cases].
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Congenital glucose-galactose malabsorption (CGGM) is a rare autosomal recessive disorder, which presents as a protracted diarrhea in early neonatal life. We describe the clinical history, diagnostic evaluation, and management of 7 children with CGGM in western France. There were 4 girls and 3 boys
Effects of oral lactose and xylose loads on blood glucose, galactose, xylose, and insulin values in healthy calves and calves with diarrhea.
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OBJECTIVE
2 hypotheses were tested: calves with acute, mild diarrhea digest lactose less efficiently than healthy calves, and they are in a catabolic state, which influences plasma glucose concentration after glucose absorption.
METHODS
Clinical study; 2 treatments with 10
[Life-threatening diarrhea in the neonatal period. A case of congenital glucose-galactose malabsorption].
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Congenital glucose-galactose malabsorption: A rare and severe cause of infant diarrhea.
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Studies on the promoting factors of galactose and lactose metabolism in the whey. IV. Concerning substances in whey that inhibit diarrhea and consequent trophopathy developed with lactose and galactose.
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[Determination of urinary galactose in infants with diarrhea].
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D28G mutation in congenital glucose-galactose malabsorption.
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BACKGROUND
Congenital glucose-galactose malabsorption is a rare autosomal recessive disorder of the intestinal transport of glucose and galactose, leading to watery diarrhea, dehydration, failure to thrive, and early death.
METHODS
In this study, we analyzed D28G mutation in 16 family members of a
Galactose epimeraseless mutants of Salmonella typhimurium as live vaccines for calves.
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The purpose of the study was to evaluate the safety and efficacy of a galactose epimeraseless mutant of Salmonella typhimurium administered as an oral vaccine to one week old calves and to investigate properties of galactose epimeraseless mutants which affect their virulence and immunogenicity. The
Nephrolithiasis in a child with glucose-galactose malabsorption.
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Glucose-galactose malabsorption (GGM) is a rare autosomal recessive disorder of intestinal transport of glucose and galactose, leading to watery diarrhea, dehydration, failure to thrive, or early death. We report a female newborn with GGM, whose clinical diagnosis was confirmed by mutational
[A new case of glucose-galactose malabsorption].
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The authors report a new case of glucose-galactose malabsorption. The particular features of this neonatal life-threatening refractory diarrhea are recalled. The dietetic management which permits a normal growth and psycho-intellectual development is described. The pathophysiological mechanisms of
Hypernatremic dehydration as a sign leading to the diagnosis of glucose-galactose malabsorption in breast-fed neonates.
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Hypernatremic dehydration is described in a case of a breast-fed infant with glucose-galactose malabsorption. Hypernatremic dehydration in general is uncommon in normal breast-fed infants. The finding of hypernatremic dehydration in breast-fed infants with watery diarrhea and sugar in the stool
SLC5A1 Mutations in Saudi Arabian Patients with Congenital Glucose-Galactose Malabsorption.
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Congenital glucose-galactose malabsorption (cGGM) is a rare autosomal recessive disorder, caused by mutations in the SLC5A1 gene, encoding the sodium/glucose cotransporter 1, which may result in severe life-threatening osmotic diarrhea due to the accumulation of unabsorbed sugars in the intestinal
Two Cases of Mistaken Polyuria and Nephrocalcinosis in Infants with Glucose-Galactose Malabsorption: A Possible Role of 1,25(OH)2D3 .
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OBJECTIVE
Glucose-galactose malabsorption (GGM) is a rare and potentially fatal disorder. The autosomal recessive mutation of the SGLT1 gene interferes with the active glucose transport in the gut resulting in osmotic diarrhea and failure to thrive (FTT). Two nonrelated infants with GGM are
[30 years' work on congenital glucose and galactose malabsorption: from phenotype to genotype].
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Intestinal absorption of glucose plays a key role in water economy as attested by the congenital and selective glucose and galactose malabsorption which is expressed as severe watery diarrhea just after birth, leading to life-threatening dehydration. This syndrome, transmitted on an autosomal
[Thirty years of research on congenital glucose and galactose malabsorption: from phenotype to genotype].
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Intestinal absorption of glucose plays a key role in water economy as attested by the congenital and selective glucose and galactose malabsorption which is expressed as severe watery diarrhea just after birth, leading to life-threatening dehydration. This syndrome, transmitted on an autosomal
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