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hyperoxaluria/carbohydrate
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Increase in calciuria and oxaluria after a single chocolate bar load.
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Chocolate, a foodstuff rich in sucrose, fat and oxalate, is considered unsuitable in cases of obesity, diabetes mellitus, urolithiasis and postprandial hypoglycemia. However the pathophysiological effects of chocolate are poorly documented. Therefore we investigated the effects of ingestion of 100 g
Dietary risk factors for hyperoxaluria in calcium oxalate stone formers.
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BACKGROUND
Hyperoxaluria is a major predisposing factor in calcium oxalate urolithiasis. The aim of the present study was to clarify the role of dietary oxalate in urinary oxalate excretion and to assess dietary risk factors for hyperoxaluria in calcium oxalate stone patients.
METHODS
Dietary
Carbohydrate metabolism and urinary excretion of calcium and oxalate after ingestion of polyol sweeteners.
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Polyols are widely used instead of glucose and sucrose in sweets and dietary products because they are barely cariogenic, and their energy value is lower. In addition, it has been shown that calciuria and oxaluria increase after an oral glucose (Glu) load. We, therefore, investigated the effects of
[Cerebrorenal oxalate formation--a metabolic abnormality following parenteral infusions of carbohydrate exchange substances].
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A 46 years old male alcoholic was admitted with an assumed alcohol withdrawal syndrome accompanied by tonic-clonic seizures. Parenteral nutrition with fructose, sorbitol, xylitol, dextran, hydroxyethylstarch, electrolytes, vitamins and amino acids was undertaken. He died 20 d later due to renal
Obesity and urolithiasis.
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The current obesity epidemic in the United States has deleterious effects on the health of the population. Temporally related to the increase in obesity is an increase in the prevalence of urolithiasis. Epidemiologic studies have shown that the incident stone risk increases with body mass index.
Gas chromatographic-mass spectrometric urinary metabolome analysis to study mutations of inborn errors of metabolism.
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Urine contains numerous metabolites, and can provide evidence for the screening or molecular diagnosis of many inborn errors of metabolism (IEMs). The metabolomic analysis of urine by the combined use of urease pretreatment, stable-isotope dilution, and capillary gas chromatography/mass spectrometry
[Hypoglycemia and endocrine effects of adults' inborn errors of metabolism].
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Inborn errors of metabolism (IEM) are rare diseases, most often inherited as an autosomal recessive disorder. They may be associated with endocrine dysfunction, the most frequent of them being disorders of carbohydrate metabolism (hypoglycemia, diabetes). The endocrinologist might be led to screen
Effect of phospholipase A2 hydrolysis products on calcium oxalate precipitation at lipid interfaces.
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Urinary stones are commonly composed of an inorganic component, calcium oxalate, or calcium phosphate and an organic matrix of lipids, carbohydrates, and proteinaceous matter. Of interest is the role that the organic matrix elements may play as catalysts for the heterogeneous nucleation of the
A comparative study of erythrocyte oxalate flux rate and urinary oxalate excretion in hyperoxaluric rats.
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The present study was carried out to investigate the relation between erythrocyte oxalate flux rate and the pathogenesis of calcium oxalate renal stone disease having hyperoxaluria as the main risk factor. Male albino rats were made hyperoxaluric by feeding them diets viz. vitamin B6 deficient,
[Physiopathology, exploration and treatment of calcium lithiasis].
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The main risk factors for calcium urolithiasis that are clinically detectable are low diuresis, hypercalciuria, hyperruricuria, alkaline urinary pH, hyperoxaluria, hypomagnesuria, hypocitraturia. They should be evaluated, all the more precisely that the disease is active, under both the urological
Patho-physiological evaluation of Duranta erecta for the treatment of urolithiasis.
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BACKGROUND
Urolithiasis is the third common disorder of the urinary system affecting 10-15% of the general population. In recent years, search for new antilithiatic drugs from natural sources has assumed greater importance.
OBJECTIVE
This study was performed to investigate the anti-urolithiatic
[Urinary excretion of calcium, magnesium, uric acid and oxalic acid in normal children].
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A group of normal children with a free diet was studied. Their first morning urine (n = 176) and their 24-hour-urine (n = 64) was collected, valuing the calciuria, magnesiuria, uricosuria and oxaluria, establishing their relationship with a dietetic survey. Ca/Cr rate value was 0.13 +/- 0.7 mg/mg in
N-glycosylation critically regulates function of oxalate transporter SLC26A6.
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The brush border Cl--oxalate exchanger SLC26A6 plays an essential role in mediating intestinal secretion of oxalate and is crucial for the maintenance of oxalate homeostasis and the prevention of hyperoxaluria and calcium oxalate nephrolithiasis. Previous in vitro studies have suggested that SLC26A6
Diet and renal stone formation.
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The relationship between diet and the formation of renal stones is demonstrated, but restrictive diets do not take into account the complexity of metabolism and the complex mechanisms that regulate the saturation and crystallization processes in the urine. The restriction of dietary calcium can
Review article: practical management of the short bowel.
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A shortened small intestine may end at a stoma or be anastomosed to the colon. Patients with a jejunostomy, but not those with a colon, lose large amounts of sodium. The intake and absorption of sodium can be increased by sipping a sodium-glucose solution; stomal loss can be reduced by restricting
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