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[Effect of the antioxidant ionol on energy metabolic indices and heart function during acute hypoxia and subsequent reoxygenation].
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The effect of preliminary administration of antioxidant ionol on the heart energy metabolism and contractile function was estimated in hypoxic hypoxia and subsequent reoxygenation. The protective effect of ionol on the energy metabolism in hypoxia was shown to occur mainly at the level of
[Effect of ionol on the activity of oxidative enzymes and ultrastructure of myocardial hematoparenchymatous barrier in hypoxic hypoxia].
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Experiments were conducted on 112 adult male Wistar rats to study the effect of preliminary ionol administration on the activity of oxidation enzymes and ultrastructure of the hematoparenchymatous barrier (HPB) in the myocardium in acute and prolonged hypoxic hypoxia. It is shown that ionol was
[Effect of the antihypoxic agent ionol on the morphofunctional state of the air-blood barrier of the lungs in hypoxic anoxia].
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It is shown that antihypoxic ionol has promoted normalization of the air-blood lung barrier ultrastructure, activation of the surfactant system under acute hypoxic hypoxia effect as well as compensatory redistribution of the thickness of separate barrier layers due to intensified synthesis of
[Effect of ionol on the hematoparenchymatous barrier of the rat myocardium in hypoxic hypoxia].
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[Correction of catecholamine-induced myocardial damage with ionol].
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Experiments on male rats exhibiting both high and low resistance to hypoxia have shown that ionol acts as a cardioprotective agent in the adrenalin-induced myocardial dystrophy. This effect is realized through the depression of the lipid peroxidation activity.
[Method of studying the effects of pharmacological substances on work capacity of animals in hypobaric hypoxia].
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The method of the study of medical agent influence and biological active substances on duration of small laboratory animals swimming has been worked out excluding the air. For this purpose the animals were placed into altitude chamber, filled with water by 1/3 (one-third) of its volume being in
[Prevention of heart damage by extreme physical stress and increasing its resistance to acute overstrain with the antioxidant ionol].
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A study of the effect of extreme physical stress on the contractility of an isolated heart, its CPK loss associated with anoxia and reoxygenation, and myocardial adenyl components and lactate levels under acute overstrain of the heart achieved through a complete obstruction of the aorta, showed the
[Lipid peroxidation and ATPase activity in synaptosomal and mitochondrial fractions of the brain in hypoxia].
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Acute hypoxia was accompanied by intensification of lipid peroxidation in synaptosomal and mitochondrial rat brain fractions as well as by inhibition of Na+, K+- and Mg2+-ATPases. Preadministration of antioxidants vitamin E and ionol into animals prevented distinctly the increase in lipid
[Lipid peroxidation in the synaptosomal and mitochondrial fractions of separate brain structures in hypoxia].
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The paper studies intensification of lipid peroxide oxidation in separate brain structures (the medulla oblongata, cerebellum, visual and sensomotor cortex), synaptosomal and mitochondrial fractions under hypoxia. It has been established that acute hypoxia increases accumulation of lipid
[The effect of hypoxia on the glucose-6-phosphate dehydrogenase activity of the erythrocytes in rats].
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Biochemical methods have shown, that adaptation of rats to the conditions of the middle mountains (2100 m above sea level) as well as with subsequent affecting acute hypoxia in the hypobaric chamber (7500 m, 2-5 h and 9000 m, 1-3 h) is accompanied by the increase of the activity of
[Prevention of stress-induced and hypoxic heart lesions by means of an antioxidant ionol].
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It was shown in experiments on male Wistar rats that activation of peroxidized oxidation of myocardial lipids and accumulation of hydroperoxides in the myocardium comprise the decisive link of the pathogenic chain of stress and hypoxic damages to the heart. Injection of a potent non-toxic
[Dissociation of the anti-amnesic and antihypoxic effects of nootropic and antihypoxic preparations].
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In experimental studies on mice it was shown that piracetam, Cleregil, centrophenoxine, pyritinol possessed the most pronounced anti-amnestic activity. A close effect was noted with Euclidan, 3-hydroxypyridine and ionol. GABAergic agents (sodium oxybutyrate, phenibut, pantogam), gutimine,
Stress damage to nonischemic divisions of the heart in experimental infarction and its prevention.
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Recently, it has been shown that nonischemic parts of the heart in myocardial infarction were separated from ischemic damaged ones by a sharp border zone. In this connection, the disturbance of contractile function of the myocardium of nonischemic parts is suggested to result from the
[Prevention of cardiac fibrillation using antioxidants and preliminary adaptation of animals to the effects of stress].
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Electrical ventricular fibrillation threshold (VFT) was determined, using programmed isolated stimuli on the left-ventricular apex, in male Wistar rats at different times after 10 hours' immobilization stress (IS). The VFT decrease reached a maximum of 48% 12 hours after IS. Cardiac sensitivity to
[Effect of emotional and pain stress on the level of antioxidant vitamins in the blood serum of rats].
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The content of ascorbic acid and alpha-tocopherol was found to be decreased in the blood of rats which had suffered an emotional pain stress. Preadaptation of the animals to hypoxia did not change the level of ascorbic acid, but it was valuable with respect to alpha-tocopherol. The emotional pain
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